Correction to “PPARα Senses Bisphenol S to Trigger EP300-Mediated Autophagy Blockage and Hepatic Steatosis”

A corrected version of Figure 1 appears below. Figure 1. Comparison of BPS levels in healthy controls and NAFLD patients. (a and b) Urine and serum level of BPS, detected by LC-MS assays. (c) Association between BPS and NAFLD in the Chinese and U.S. participants. (d) Association between urine BPS and lipid levels and liver enzymes. (e) Association between serum BPS and lipid levels and liver enzymes. ^aModel adjusted for age, gender, race, education level, household income level, smoking exposure, urinary creatinine, log-transformed levels of TG and HDL, and log-transformed concentration of BPA in U.S. participants. ^bModel adjusted for age, gender, log-transformed levels of TG and HDL, and log-transformed concentration of BPA in Chinese participants. ^cModel adjusted for BMI, age, gender, education level, smoking exposure, log-transformed levels of TG and HDL, and log-transformed concentration of BPA in Chinese participants. In addition, the following sentences from lines 3–7 of the first paragraph of section 3.1 of Results have been corrected. “The average concentration of serum BPS in mice was 0.45 ± 0.28 ng/mL. The NAFLD activity score and the ratio of liver and body weight were significantly higher in the BPS group (Figure 2b,c). BPS exposure significantly increased TG in liver and serum (Figure 2d,e and Figure S2a–d).” This article has not yet been cited by other publications.