细胞融合影响p53动态响应DNA损伤。

IF 3.1 3区 生物学 Q3 CELL BIOLOGY
Molecular Biology of the Cell Pub Date : 2025-06-01 Epub Date: 2025-04-09 DOI:10.1091/mbc.E24-09-0394
Alina Simerzin, Emily E Ackerman, Kotaro Fujimaki, Rainer H Kohler, Yoshiko Iwamoto, Mathias S Heltberg, Ashwini Jambhekar, Ralph Weissleder, Galit Lahav
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引用次数: 0

摘要

肿瘤抑制蛋白p53在细胞对DNA损伤的反应中起着关键作用。在DNA双链断裂(DSB)的反应中,培养细胞表现出p53水平的振荡,从而影响基因表达和细胞命运。p53在体内的动态只在固定组织中或使用p53的转录活性报告进行了研究。在这里,我们建立了表达p53水平荧光报告基因的乳腺肿瘤,并采用活体成像来量化其在体内对DSB的反应动态。我们的发现揭示了单个细胞之间的巨大异质性,大多数细胞表现出单一的延长脉冲。然后,我们测试了p53动力学如何在高细胞融合下发生变化,这是细胞培养和组织之间不同的一个因素。我们发现高度融合培养的乳腺癌细胞也显示一个宽的p53脉冲而不是振荡。通过数学建模、敏感性分析和活细胞成像,我们确定了低水平的磷酸酶Wip1 (p53的转录靶点和负调节因子)是这些动态的关键因素。由于高细胞融合性更好地反映了组织的微环境,细胞融合性对p53动力学的影响可能对癌组织对DNA损伤诱导疗法的反应具有重要影响。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Cell confluency affects p53 dynamics in response to DNA damage.

The tumor suppressor protein p53 plays a key role in the cellular response to DNA damage. In response to DNA double-strand breaks (DSB), cultured cells exhibit oscillations of p53 levels, which impact gene expression and cell fate. The dynamics of p53 in vivo have only been studied in fixed tissues or using reporters for p53's transcriptional activity. Here we established breast tumors expressing a fluorescent reporter for p53 levels and employed intravital imaging to quantify its dynamics in response to DSB in vivo. Our findings revealed large heterogeneity among individual cells, with most cells exhibiting a single prolonged pulse. We then tested how p53 dynamics might change under high cell confluency, one factor that differs between cell culture and tissues. We revealed that highly confluent cultured breast cancer cells also show one broad p53 pulse instead of oscillations. Through mathematical modeling, sensitivity analysis, and live-cell imaging, we identified low levels of the phosphatase Wip1, a transcriptional target and negative regulator of p53, as a key contributor to these dynamics. Because high cell confluency better reflects the microenvironment of tissues, the impact of cell confluency on p53 dynamics may have important consequences for cancerous tissues responding to DNA damage-inducing therapies.

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来源期刊
Molecular Biology of the Cell
Molecular Biology of the Cell 生物-细胞生物学
CiteScore
6.00
自引率
6.10%
发文量
402
审稿时长
2 months
期刊介绍: MBoC publishes research articles that present conceptual advances of broad interest and significance within all areas of cell, molecular, and developmental biology. We welcome manuscripts that describe advances with applications across topics including but not limited to: cell growth and division; nuclear and cytoskeletal processes; membrane trafficking and autophagy; organelle biology; quantitative cell biology; physical cell biology and mechanobiology; cell signaling; stem cell biology and development; cancer biology; cellular immunology and microbial pathogenesis; cellular neurobiology; prokaryotic cell biology; and cell biology of disease.
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