{"title":"对“神经调节蛋白4通过激活AMPK/ mtor介导的小鼠自噬,部分减轻足细胞损伤和蛋白尿”的更正","authors":"","doi":"10.1002/jcb.70034","DOIUrl":null,"url":null,"abstract":"<p>J. Deng, Q. Yang, W. Zhu, et al., “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice,” <i>Journal of Cellular Biochemistry</i> 125, no. 10 (2024): e30634. https://doi.org/10.1002/jcb.30634.</p><p>The authors realized that there was an error in the results of the Nephrin immunofluorescence staining of NRG4 group in Figure 6D. The correct Figure 6 with the modified NRG4 group is shown below. This correction does not change the results and conclusions of the manuscript.</p><p>We apologize for this error.</p>","PeriodicalId":15219,"journal":{"name":"Journal of cellular biochemistry","volume":"126 5","pages":""},"PeriodicalIF":3.0000,"publicationDate":"2025-05-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcb.70034","citationCount":"0","resultStr":"{\"title\":\"Correction to “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice”\",\"authors\":\"\",\"doi\":\"10.1002/jcb.70034\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>J. Deng, Q. Yang, W. Zhu, et al., “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice,” <i>Journal of Cellular Biochemistry</i> 125, no. 10 (2024): e30634. https://doi.org/10.1002/jcb.30634.</p><p>The authors realized that there was an error in the results of the Nephrin immunofluorescence staining of NRG4 group in Figure 6D. The correct Figure 6 with the modified NRG4 group is shown below. This correction does not change the results and conclusions of the manuscript.</p><p>We apologize for this error.</p>\",\"PeriodicalId\":15219,\"journal\":{\"name\":\"Journal of cellular biochemistry\",\"volume\":\"126 5\",\"pages\":\"\"},\"PeriodicalIF\":3.0000,\"publicationDate\":\"2025-05-05\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/jcb.70034\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Journal of cellular biochemistry\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/jcb.70034\",\"RegionNum\":3,\"RegionCategory\":\"生物学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q3\",\"JCRName\":\"BIOCHEMISTRY & MOLECULAR BIOLOGY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Journal of cellular biochemistry","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/jcb.70034","RegionNum":3,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q3","JCRName":"BIOCHEMISTRY & MOLECULAR BIOLOGY","Score":null,"Total":0}
Correction to “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice”
J. Deng, Q. Yang, W. Zhu, et al., “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice,” Journal of Cellular Biochemistry 125, no. 10 (2024): e30634. https://doi.org/10.1002/jcb.30634.
The authors realized that there was an error in the results of the Nephrin immunofluorescence staining of NRG4 group in Figure 6D. The correct Figure 6 with the modified NRG4 group is shown below. This correction does not change the results and conclusions of the manuscript.
期刊介绍:
The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.