对“神经调节蛋白4通过激活AMPK/ mtor介导的小鼠自噬,部分减轻足细胞损伤和蛋白尿”的更正

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

摘要

邓杰,杨琪,朱伟,等,“神经调节蛋白4在一定程度上通过激活AMPK/ mmir介导的自噬来减轻小鼠足细胞损伤和蛋白尿,”细胞生物化学杂志,第125期。10 (2024): e30634。https://doi.org/10.1002/jcb.30634.The作者意识到图6D中NRG4组的Nephrin免疫荧光染色结果存在误差。修改后的NRG4组的正确图6如下所示。此更正不改变稿件的结果和结论。我们为这个错误道歉。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Correction to “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice”

Correction to “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice”

J. Deng, Q. Yang, W. Zhu, et al., “Neuregulin 4 Attenuates Podocyte Injury and Proteinuria in Part by Activating AMPK/mTOR-Mediated Autophagy in Mice,” Journal of Cellular Biochemistry 125, no. 10 (2024): e30634. https://doi.org/10.1002/jcb.30634.

The authors realized that there was an error in the results of the Nephrin immunofluorescence staining of NRG4 group in Figure 6D. The correct Figure 6 with the modified NRG4 group is shown below. This correction does not change the results and conclusions of the manuscript.

We apologize for this error.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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