{"title":"更正“Ta4C3纳米片作为光热驱动的ROS清除和免疫激活对抗糖尿病伤口抗生素耐药感染的新治疗平台”","authors":"Yapeng Wang, Jing Yang, Yunhong Ma, Jun Liu, Peng Wang, Junhao Luo, Yongjun Rui, Yongwei Wu","doi":"10.1002/smll.202502209","DOIUrl":null,"url":null,"abstract":"<p><i>small</i>, <b>2024</b>, <i>20</i>, 2400741</p>\n<p>DOI: 10.1002/smll.202400741</p>\n<p>We sincerely acknowledge an error in the originally published Figure 5i (Ta₄C₃ NSs + NIR group). The correct image is provided below. We confirm that this correction does not affect the scientific conclusions or overall interpretation of the study. We apologize for any confusion this may have caused and appreciate the readers' understanding:</p>\n<p><img alt=\"image\" loading=\"lazy\" src=\"/cms/asset/e88913fb-daa9-47cd-a309-957ff11d8053/smll202502209-gra-0001.png\"/></p>\n<p>Figure 5. Assessing Immune Memory Triggered by Ta<sub>4</sub>C<sub>3</sub> NSs and NIR in a Recurrent MRSA Infection Model. A) Illustrates the development of the abscess model and the treatment strategy. On Day 30, the concentrations of serum interleukins IL-4 B), IFN𝛾 C), IL-17 D), and TNF-𝛼 E) were quantified (mean ± SEM, <i>n</i> = 5). F) To evaluate memory B (mB) cells, blood single-cell suspensions were subjected to flow cytometry (FCM) analysis following staining with CD45, CD31, and CD19 antibodies (mean ± SEM, <i>n</i> = 5). G–J) The study included an assessment of CD45+ B220+ memory B cells in lymph nodes (G,H) and spleens (I,J) of mice across different treatment groups on Day 30 (mean ± SEM, <i>n</i> = 5). K,L) The formation of MRSA colonies on LB-agar plates K) and the tally of surviving bacteria L) were documented on day 30. M) Conceptual representation of the mechanism by which Ta<sub>4</sub>C<sub>3</sub> NSs prevent recurrent MRSA infections. Significance levels are marked as ns (not significant), <sup>*</sup><i>p</i> < 0.05, <sup>**</sup><i>p</i> < 0.01, <sup>***</sup><i>p</i> < 0.001, and <sup>****</sup><i>p</i> < 0.0001, as determined by one-way ANOVA.</p>\n<p>We apologize for this error.</p>","PeriodicalId":228,"journal":{"name":"Small","volume":"15 1","pages":""},"PeriodicalIF":13.0000,"publicationDate":"2025-04-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Correction to “Ta4C3 Nanosheets as a Novel Therapeutic Platform for Photothermal-Driven ROS Scavenging and Immune Activation Against Antibiotic-Resistant Infections in Diabetic Wounds”\",\"authors\":\"Yapeng Wang, Jing Yang, Yunhong Ma, Jun Liu, Peng Wang, Junhao Luo, Yongjun Rui, Yongwei Wu\",\"doi\":\"10.1002/smll.202502209\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><i>small</i>, <b>2024</b>, <i>20</i>, 2400741</p>\\n<p>DOI: 10.1002/smll.202400741</p>\\n<p>We sincerely acknowledge an error in the originally published Figure 5i (Ta₄C₃ NSs + NIR group). The correct image is provided below. We confirm that this correction does not affect the scientific conclusions or overall interpretation of the study. We apologize for any confusion this may have caused and appreciate the readers' understanding:</p>\\n<p><img alt=\\\"image\\\" loading=\\\"lazy\\\" src=\\\"/cms/asset/e88913fb-daa9-47cd-a309-957ff11d8053/smll202502209-gra-0001.png\\\"/></p>\\n<p>Figure 5. Assessing Immune Memory Triggered by Ta<sub>4</sub>C<sub>3</sub> NSs and NIR in a Recurrent MRSA Infection Model. A) Illustrates the development of the abscess model and the treatment strategy. On Day 30, the concentrations of serum interleukins IL-4 B), IFN𝛾 C), IL-17 D), and TNF-𝛼 E) were quantified (mean ± SEM, <i>n</i> = 5). F) To evaluate memory B (mB) cells, blood single-cell suspensions were subjected to flow cytometry (FCM) analysis following staining with CD45, CD31, and CD19 antibodies (mean ± SEM, <i>n</i> = 5). G–J) The study included an assessment of CD45+ B220+ memory B cells in lymph nodes (G,H) and spleens (I,J) of mice across different treatment groups on Day 30 (mean ± SEM, <i>n</i> = 5). K,L) The formation of MRSA colonies on LB-agar plates K) and the tally of surviving bacteria L) were documented on day 30. M) Conceptual representation of the mechanism by which Ta<sub>4</sub>C<sub>3</sub> NSs prevent recurrent MRSA infections. Significance levels are marked as ns (not significant), <sup>*</sup><i>p</i> < 0.05, <sup>**</sup><i>p</i> < 0.01, <sup>***</sup><i>p</i> < 0.001, and <sup>****</sup><i>p</i> < 0.0001, as determined by one-way ANOVA.</p>\\n<p>We apologize for this error.</p>\",\"PeriodicalId\":228,\"journal\":{\"name\":\"Small\",\"volume\":\"15 1\",\"pages\":\"\"},\"PeriodicalIF\":13.0000,\"publicationDate\":\"2025-04-25\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Small\",\"FirstCategoryId\":\"88\",\"ListUrlMain\":\"https://doi.org/10.1002/smll.202502209\",\"RegionNum\":2,\"RegionCategory\":\"材料科学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"CHEMISTRY, MULTIDISCIPLINARY\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Small","FirstCategoryId":"88","ListUrlMain":"https://doi.org/10.1002/smll.202502209","RegionNum":2,"RegionCategory":"材料科学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"CHEMISTRY, MULTIDISCIPLINARY","Score":null,"Total":0}
Correction to “Ta4C3 Nanosheets as a Novel Therapeutic Platform for Photothermal-Driven ROS Scavenging and Immune Activation Against Antibiotic-Resistant Infections in Diabetic Wounds”
small, 2024, 20, 2400741
DOI: 10.1002/smll.202400741
We sincerely acknowledge an error in the originally published Figure 5i (Ta₄C₃ NSs + NIR group). The correct image is provided below. We confirm that this correction does not affect the scientific conclusions or overall interpretation of the study. We apologize for any confusion this may have caused and appreciate the readers' understanding:
Figure 5. Assessing Immune Memory Triggered by Ta4C3 NSs and NIR in a Recurrent MRSA Infection Model. A) Illustrates the development of the abscess model and the treatment strategy. On Day 30, the concentrations of serum interleukins IL-4 B), IFN𝛾 C), IL-17 D), and TNF-𝛼 E) were quantified (mean ± SEM, n = 5). F) To evaluate memory B (mB) cells, blood single-cell suspensions were subjected to flow cytometry (FCM) analysis following staining with CD45, CD31, and CD19 antibodies (mean ± SEM, n = 5). G–J) The study included an assessment of CD45+ B220+ memory B cells in lymph nodes (G,H) and spleens (I,J) of mice across different treatment groups on Day 30 (mean ± SEM, n = 5). K,L) The formation of MRSA colonies on LB-agar plates K) and the tally of surviving bacteria L) were documented on day 30. M) Conceptual representation of the mechanism by which Ta4C3 NSs prevent recurrent MRSA infections. Significance levels are marked as ns (not significant), *p < 0.05, **p < 0.01, ***p < 0.001, and ****p < 0.0001, as determined by one-way ANOVA.
期刊介绍:
Small serves as an exceptional platform for both experimental and theoretical studies in fundamental and applied interdisciplinary research at the nano- and microscale. The journal offers a compelling mix of peer-reviewed Research Articles, Reviews, Perspectives, and Comments.
With a remarkable 2022 Journal Impact Factor of 13.3 (Journal Citation Reports from Clarivate Analytics, 2023), Small remains among the top multidisciplinary journals, covering a wide range of topics at the interface of materials science, chemistry, physics, engineering, medicine, and biology.
Small's readership includes biochemists, biologists, biomedical scientists, chemists, engineers, information technologists, materials scientists, physicists, and theoreticians alike.