缩回：叉头盒p3通过调控MicroRNA-146a调控口腔扁平苔藓的进展

IF 3 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of cellular biochemistry Pub Date : 2025-04-23 DOI:10.1002/jcb.70019

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引用次数: 0

摘要

退稿：J. Wang, L. Yang, L. Wang, Y. Yang, and Y. Wang, "Forkhead Box p3 Controls Progression of Oral Lichen Planus by Regulating MicroRNA-146a, " Journal of Cellular Biochemistry 119, no：8862-8871, https://doi.org/10.1002/jcb.27139.The 上述文章于 2018 年 8 月 20 日在线发表于 Wiley Online Library (wileyonlinelibrary.com)，经期刊主编 Christian Behl 和 Wiley Periodicals LLC 协议，该文章已被撤回。同意撤稿的原因是第三方提出的疑虑。图 2E、3D 和 5 A 中的图像元素被发现曾由不同作者在不同科学背景下发表过。我们邀请作者就这些问题发表评论，但他们没有回应。因此，由于编者对文章中提供的全部数据的完整性和可靠性失去信心，并认为其结论无效，文章被撤回。已将撤稿通知作者。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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RETRACTION: Forkhead Box p3 Controls Progression of Oral Lichen Planus by Regulating MicroRNA-146a

RETRACTION: J. Wang, L. Yang, L. Wang, Y. Yang, and Y. Wang, “Forkhead Box p3 Controls Progression of Oral Lichen Planus by Regulating MicroRNA-146a, ” Journal of Cellular Biochemistry 119, no. 11 (2018): 8862-8871, https://doi.org/10.1002/jcb.27139.

The above article, published online on 20 August 2018 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed due to concerns raised by third parties. Image elements within Figures 2E, 3D and 5 A were found to have been published previously by different authors in different scientific contexts. The authors were invited to comment on these concerns but did not respond. Accordingly, the article is retracted as the editors have lost confidence in the integrity and reliability of the full body of data presented in the article and consider its conclusions invalid. The authors were informed of the retraction.

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.