NUP62通过依赖nsd2的表观遗传重编程,减轻人牙髓干细胞的衰老并促进其干性

IF 10.8 1区 医学 Q1 DENTISTRY, ORAL SURGERY & MEDICINE
Xiping Wang, Li Wang, Linxi Zhou, Lu Chen, Jiayi Shi, Jing Ge, Sha Tian, Zihan Yang, Yuqiong Zhou, Qihao Yu, Jiacheng Jin, Chen Ding, Yihuai Pan, Duohong Zou
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引用次数: 0

摘要

干细胞在维持组织再生能力和体内平衡中起着至关重要的作用。然而,与干细胞衰老相关的机制需要进一步研究。在这项研究中,我们对来自不同年龄个体的人牙髓干细胞(HDPSCs)进行了蛋白质组学分析。我们的研究结果表明,NUP62在衰老的hdpsc中表达降低。我们发现NUP62在体外和体内均能减轻衰老相关表型并增强分化潜力。相反,NUP62表达的下调加重了衰老相关表型,损害了hdpsc的增殖和迁移能力。通过rna测序和解码NUP62过表达诱导的表观基因组景观,我们发现NUP62通过增强转录因子E2F1的核转运来帮助减缓hdpsc的衰老。这反过来又刺激表观遗传酶NSD2的转录。最后,NUP62的过表达影响抗衰老基因(HMGA1、HMGA2和SIRT6)的H3K36me2和H3K36me3修饰。我们的研究结果表明,NUP62通过依赖nsd2的表观遗传重编程调节hdpsc的命运。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

NUP62 alleviates senescence and promotes the stemness of human dental pulp stem cells via NSD2-dependent epigenetic reprogramming

NUP62 alleviates senescence and promotes the stemness of human dental pulp stem cells via NSD2-dependent epigenetic reprogramming

Stem cells play a crucial role in maintaining tissue regenerative capacity and homeostasis. However, mechanisms associated with stem cell senescence require further investigation. In this study, we conducted a proteomic analysis of human dental pulp stem cells (HDPSCs) obtained from individuals of various ages. Our findings showed that the expression of NUP62 was decreased in aged HDPSCs. We discovered that NUP62 alleviated senescence-associated phenotypes and enhanced differentiation potential both in vitro and in vivo. Conversely, the knocking down of NUP62 expression aggravated the senescence-associated phenotypes and impaired the proliferation and migration capacity of HDPSCs. Through RNA-sequence and decoding the epigenomic landscapes remodeled induced by NUP62 overexpression, we found that NUP62 helps alleviate senescence in HDPSCs by enhancing the nuclear transport of the transcription factor E2F1. This, in turn, stimulates the transcription of the epigenetic enzyme NSD2. Finally, the overexpression of NUP62 influences the H3K36me2 and H3K36me3 modifications of anti-aging genes (HMGA1, HMGA2, and SIRT6). Our results demonstrated that NUP62 regulates the fate of HDPSCs via NSD2-dependent epigenetic reprogramming.

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来源期刊
International Journal of Oral Science
International Journal of Oral Science DENTISTRY, ORAL SURGERY & MEDICINE-
CiteScore
31.80
自引率
1.30%
发文量
53
审稿时长
>12 weeks
期刊介绍: The International Journal of Oral Science covers various aspects of oral science and interdisciplinary fields, encompassing basic, applied, and clinical research. Topics include, but are not limited to: Oral microbiology Oral and maxillofacial oncology Cariology Oral inflammation and infection Dental stem cells and regenerative medicine Craniofacial surgery Dental material Oral biomechanics Oral, dental, and maxillofacial genetic and developmental diseases Craniofacial bone research Craniofacial-related biomaterials Temporomandibular joint disorder and osteoarthritis The journal publishes peer-reviewed Articles presenting new research results and Review Articles offering concise summaries of specific areas in oral science.
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