更正“羟基红花黄A通过Nrf2/HO‐1和炎症信号通路减弱卵清蛋白诱导的变应性鼻炎的过敏反应”

IF 3.2 3区医学 Q2 ENVIRONMENTAL SCIENCES

Environmental Toxicology Pub Date : 2025-04-16 DOI:10.1002/tox.24518

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引用次数: 0

摘要

马庆，李国光，何建军，王建军，叶波。“羟基红花黄A通过Nrf2/HO-1和炎症信号通路减轻卵白蛋白诱导的变应性鼻炎的过敏反应”，《环境毒理学》38，no。7 (2023): 1520-1534, https://doi.org/10.1002/tox.23783.This文章的通讯作者Ben Ye被错误地链接到两个附属机构：中国淄博市第九人民医院耳鼻咽喉科和中国济南山东第一医科大学附属山东省医院耳鼻咽喉科。本叶正确且唯一的所属单位应为山东第一医科大学附属山东省医院耳鼻咽喉科，中国济南。这已经在文章中更正了。我们为这个错误道歉。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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Correction to “Hydroxysafflor Yellow A Attenuates Allergic Response of Ovalbumin Induced Allergic Rhinitis via Nrf2/HO-1 and Inflammatory Signaling Pathways”

Q. Ma, G. Li, J. He, J. Wang, and B. Ye. “Hydroxysafflor Yellow A Attenuates Allergic Response of Ovalbumin Induced Allergic Rhinitis via Nrf2/HO-1 and Inflammatory Signaling Pathways,” Environmental Toxicology 38, no. 7 (2023): 1520–1534, https://doi.org/10.1002/tox.23783.

This article published with corresponding author Ben Ye being linked incorrectly to two affiliations, Department of Otorhinolaryngology, Zibo Ninth People's Hospital, Zibo, China and Department of Otorhinolaryngology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, China.

The correct and only affiliation for Ben Ye should be Department of Otorhinolaryngology, Shandong Provincial Hospital Affiliated to Shandong First Medical University, Jinan, China.

This has been corrected on the article.

We apologize for this error.

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来源期刊

Environmental Toxicology 环境科学-毒理学

CiteScore

7.10

自引率

8.90%

发文量

261

审稿时长

4.5 months

期刊介绍： The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.