撤回:lncRNA NEAT1/miR-493-5p/Rab27A轴在溃疡性结肠炎中的表达及其重要作用

IF 3.1 4区 医学 Q3 IMMUNOLOGY
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引用次数: 0

摘要

引用本文:王慧,邓军,王敏,张艳,刘晓霞,刘忠,“NEAT1/miR-493-5p/Rab27A轴在溃疡性结肠炎中的表达及其意义”,《免疫与炎症杂志》,第11期,no。5 (2023): e814, https://doi.org/10.1002/iid3.814.The上述文章于2023年5月16日在线发表在Wiley online Library (wileyonlinelibrary.com)上,经作者同意撤回;杂志主编Marc Veldhoen;约翰·威利&;子有限公司在对第三方提出的问题进行调查后,双方同意撤回这篇文章。研究发现,图5E所示的LPS+lncRNA NEAT1-sRNA+抑制剂对照流式细胞术图和部分LPS图已发表在代表不同科学背景的其他文章中。作者提供了一个解释和一些数据,但这被认为是不够的。编辑们对所提供的数据失去了信心,认为结论有问题。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Expression and Significant Roles of the lncRNA NEAT1/miR-493-5p/Rab27A Axis in Ulcerative Colitis

RETRACTION: H. Wang, J. Teng, M. Wang, Y. Zhang, X. Liu and Z. Liu, “Expression and Significant Roles of the lncRNA NEAT1/miR-493-5p/Rab27A Axis in Ulcerative Colitis,” Immunity, Inflammation and Disease 11, no. 5 (2023): e814, https://doi.org/10.1002/iid3.814.

The above article, published online on 16 May 2023 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Marc Veldhoen; and John Wiley & Sons Ltd. The retraction has been agreed upon following an investigation into concerns raised by a third party. The investigation revealed that the LPS+lncRNA NEAT1-sRNA+inhibitor control flow cytometry plot and parts of the LPS plot presented in Figure 5E have been published in other articles representing different scientific contexts. The authors provided an explanation and some data, but this was not deemed sufficient. The editors have lost confidence in the data presented and consider the conclusions to be compromised.

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来源期刊
Immunity, Inflammation and Disease
Immunity, Inflammation and Disease Medicine-Immunology and Allergy
CiteScore
3.60
自引率
0.00%
发文量
146
审稿时长
8 weeks
期刊介绍: Immunity, Inflammation and Disease is a peer-reviewed, open access, interdisciplinary journal providing rapid publication of research across the broad field of immunology. Immunity, Inflammation and Disease gives rapid consideration to papers in all areas of clinical and basic research. The journal is indexed in Medline and the Science Citation Index Expanded (part of Web of Science), among others. It welcomes original work that enhances the understanding of immunology in areas including: • cellular and molecular immunology • clinical immunology • allergy • immunochemistry • immunogenetics • immune signalling • immune development • imaging • mathematical modelling • autoimmunity • transplantation immunology • cancer immunology
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