{"title":"修正“脑内皮细胞衰老介导衰老血脑屏障功能障碍”。","authors":"","doi":"10.1111/acel.70067","DOIUrl":null,"url":null,"abstract":"<p>\n <span>Novo, J. P.</span>, <span>Gee, L.</span>, <span>Caetano, C. A.</span>, <span>Tomé, I.</span>, <span>Vilaça, A.</span>, <span>Zglinicki, T.</span>, <span>Moreira, I. S.</span>, <span>Jurk, D.</span>, <span>Rosa, S.</span>, & <span>Ferreira, L.</span> (<span>2024</span>). <span>Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence</span>. <i>Aging Cell</i>, <span>23</span>, e14270. https://doi.org/10.1111/acel.14270\n </p><p>In the published version of the above article, we noticed a few typographical errors in Figure 1a(III) and in Figure 2.</p><p>The corrected figures are shown below:</p><p>We apologize for this error.</p>","PeriodicalId":55543,"journal":{"name":"Aging Cell","volume":"24 5","pages":""},"PeriodicalIF":7.8000,"publicationDate":"2025-04-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/acel.70067","citationCount":"0","resultStr":"{\"title\":\"Correction to “Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence”\",\"authors\":\"\",\"doi\":\"10.1111/acel.70067\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>\\n <span>Novo, J. P.</span>, <span>Gee, L.</span>, <span>Caetano, C. A.</span>, <span>Tomé, I.</span>, <span>Vilaça, A.</span>, <span>Zglinicki, T.</span>, <span>Moreira, I. S.</span>, <span>Jurk, D.</span>, <span>Rosa, S.</span>, & <span>Ferreira, L.</span> (<span>2024</span>). <span>Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence</span>. <i>Aging Cell</i>, <span>23</span>, e14270. https://doi.org/10.1111/acel.14270\\n </p><p>In the published version of the above article, we noticed a few typographical errors in Figure 1a(III) and in Figure 2.</p><p>The corrected figures are shown below:</p><p>We apologize for this error.</p>\",\"PeriodicalId\":55543,\"journal\":{\"name\":\"Aging Cell\",\"volume\":\"24 5\",\"pages\":\"\"},\"PeriodicalIF\":7.8000,\"publicationDate\":\"2025-04-07\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/acel.70067\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Aging Cell\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/acel.70067\",\"RegionNum\":1,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"Biochemistry, Genetics and Molecular Biology\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Aging Cell","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/acel.70067","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"Biochemistry, Genetics and Molecular Biology","Score":null,"Total":0}
引用次数: 0
摘要
Novo, j.p, Gee, L., Caetano, C. A., tom, I., vilaa, A., Zglinicki, T., Moreira, I. S., Jurk, D., Rosa, S., &;费雷拉,L.(2024)。衰老时血脑屏障功能障碍是由脑内皮衰老介导的。衰老细胞,23,e14270。https://doi.org/10.1111/acel.14270在上述文章的发布版本中,我们注意到图1a(III)和图2中的一些排版错误。更正后的数字如下:我们为这个错误道歉。
Correction to “Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence”
Novo, J. P., Gee, L., Caetano, C. A., Tomé, I., Vilaça, A., Zglinicki, T., Moreira, I. S., Jurk, D., Rosa, S., & Ferreira, L. (2024). Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence. Aging Cell, 23, e14270. https://doi.org/10.1111/acel.14270
In the published version of the above article, we noticed a few typographical errors in Figure 1a(III) and in Figure 2.
期刊介绍:
Aging Cell, an Open Access journal, delves into fundamental aspects of aging biology. It comprehensively explores geroscience, emphasizing research on the mechanisms underlying the aging process and the connections between aging and age-related diseases.