撤回:牙龈卟啉单胞菌和牙干细胞串扰放大牙周炎生态位的炎症和骨质流失

IF 4.5 2区 生物学 Q2 CELL BIOLOGY
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引用次数: 0

摘要

引用本文:张志强,张志强,“牙龈卟啉单胞菌和牙干细胞的相互作用增强了牙周炎生态位的炎症和骨质流失”,《中华口腔医学杂志》,第2期。10 (2022): 3768-3777, https://doi.org/10.1002/jcp.30848.The上述文章于2022年8月4日在线发表在Wiley online Library (wileyonlinelibrary.com)上,经主编Robert Heath同意撤回;和Wiley期刊有限责任公司。在发表之后,该杂志注意到,这篇文章完全是基于折衷的同行评议过程而被接受的。因此,作出了撤回这一条款的决定。作者已被告知撤稿决定,不同意撤稿决定。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Porphyromonas Gingivalis and Dental Stem Cells Crosstalk Amplify Inflammation and Bone Loss in the Periodontitis Niche

RETRACTION: D. Ezhilarasan, and S. S. Varghese, “Porphyromonas Gingivalis and Dental Stem Cells Crosstalk Amplify Inflammation and Bone Loss in the Periodontitis Niche,” Journal of Cellular Physiology 237, no. 10 (2022): 3768-3777, https://doi.org/10.1002/jcp.30848.

The above article, published online on 4 August 2022 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. Following publication, it has come to the attention of the journal that the article was accepted solely on the basis of compromised peer review processes. Therefore, the decision to retract this article was taken. The authors have been informed of the decision of retraction and disagree with it.

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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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