抑制单核细胞样树突状细胞(MCs)调节呼吸道CD8+ t细胞免疫。

IF 3.6 3区 医学 Q2 IMMUNOLOGY
Katie L Reagin, Kimberly E Oliva, Matthew R Hansen, Chris D Slade, Wendy T Watford, Kimberly D Klonowski
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引用次数: 0

摘要

主动免疫抑制可以调节保护性细胞免疫和有害免疫病理之间的平衡。这种抑制可能发生在局部,感染部位或区域引流淋巴结(dln)。免疫调节在肺等部位尤其重要,异常的免疫病理可导致组织功能丧失和呼吸衰竭。我们最近在体外发现了一种新的CD11b+CD103+CCR2+单核细胞样树突状细胞(MCs),它直接抑制CD8+ t细胞的增殖。感染RNA病毒的小鼠呼吸道感染将这些MCs招募到dLN(在水泡性口炎病毒感染后)或dLN和病毒复制部位(在流感感染后)。本研究表明,使用CCR2-DTR骨髓嵌合体从小鼠dLN中去除MCs可增强呼吸道CD8+ t细胞反应和肺组织常驻记忆细胞(TRM)的形成,这与外源VSV攻击时增强抗病毒反应相关。相反,流感感染后dLN和呼吸道MCs的消耗导致呼吸道CD8+ t细胞反应增强,并伴有致命的免疫病理。总之,这些数据表明,抑制性MCs控制呼吸道CD8+ t细胞免疫的关键方面,从而在病毒感染的背景下平衡免疫和不良病理。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Regulation of respiratory CD8+ T-cell immunity by suppressive monocyte-like dendritic cells (MCs).

Active immune suppression can mediate the balance between protective cellular immunity and harmful immunopathology. This suppression can occur locally, at an infection site, or in regional draining lymph nodes (dLNs). Immune regulation is of particular importance in sites such as the lung where aberrant immunopathology can result in loss of tissue function and respiratory failure. We have recently identified a novel population of CD11b+CD103+CCR2+ monocyte-like dendritic cells (MCs) which directly suppress CD8+ T-cell proliferation in vitro. Respiratory infection of mice with RNA viruses recruits these MCs either exclusively to the dLN (after vesicular stomatitis virus infection) or both the dLN and site of viral replication (after influenza infection). Here we show that depletion of MCs from the dLN of mice using CCR2-DTR bone marrow chimeras results in enhanced respiratory CD8+ T-cell responses and lung tissue-resident memory cell (TRM) formation which correlated with enhanced antiviral responses upon heterologous VSV challenge. Conversely, depletion of MCs from both the dLN and respiratory tract following influenza infection results in enhanced respiratory CD8+ T-cell responses coupled with fatal immunopathology. Together, these data suggest that suppressive MCs govern key aspects of respiratory CD8+ T-cell immunity, thereby balancing immunity and adverse pathology in the context of viral infection.

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来源期刊
Journal of immunology
Journal of immunology 医学-免疫学
CiteScore
8.20
自引率
2.30%
发文量
495
审稿时长
1 months
期刊介绍: The JI publishes novel, peer-reviewed findings in all areas of experimental immunology, including innate and adaptive immunity, inflammation, host defense, clinical immunology, autoimmunity and more. Special sections include Cutting Edge articles, Brief Reviews and Pillars of Immunology. The JI is published by The American Association of Immunologists (AAI)
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