Sebastian Skoczylas, Tomasz Płoszaj, Karolina Gadzalska, Monika Gorządek, Paulina Jakiel, Ewa Juścińska, Maria Malarska, Magdalena Traczyk-Borszyńska, Hanna Biezynska, Magdalena Rychlik, Agata Pastorczak, Agnieszka Zmysłowska
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引用次数: 0
摘要
线粒体 DNA 变异在多种疾病中起着至关重要的作用,但由于线粒体 DNA 变异的异质性、与年龄相关的变体丢失和表型的演变,解释线粒体 DNA 变异的临床意义具有挑战性。本研究的目的是识别线粒体致病变体,并探索已知变体和新型变体未来与患者一生中特定表型的潜在关联。我们使用 Python 管道分析了 418 名患者(中位年龄:15 岁;52.9% 为男性,47.1% 为女性)的外显子组测序数据,这些患者大多被诊断为神经系统疾病、发育和智力障碍、行为和感官障碍、心血管和代谢异常、肾脏疾病等。筛查发现了 1,000 个异质性水平超过 10%的独特变异体,以及 192 个异质性水平在 1-10% 之间的独特变异体(不包括高变异区)。在这些变异中,根据 MITOMAP 发现了 4 个确定的致病变异(m.1555 A > G、m.3243 A > G、m.9035T > C 和 m.11778G > A),每个变异都在一名患者中发现。通过应用致病性和频率标准,在单卵双生姐妹中发现了三个独特的变异体和一个低异质性变异体,并通过下一代测序进行了确认。最后,其中一个变体 m.15897G > A 被确认为可能致病(PP3、PS2)。我们的研究凸显了诊断与 mtDNA 突变相关的线粒体疾病的复杂性,并强调需要采用综合的基因型-表型方法来正确识别致病变异。
Mitochondrial DNA variants revealed by whole exome sequencing: from screening to diagnosis and follow-up.
Mutations in mitochondrial DNA play a crucial role in several diseases, but interpreting the clinical significance of mitochondrial DNA variants is challenging due to heteroplasmy, age-related loss of variants and evolving phenotypes. The aim of study was to identify mitochondrial pathogenic variants and explore their potential future association with specific phenotypes in patients during their lifetime, for both known and novel variants. We used a Python pipeline to analyse exome sequencing data from 418 patients (median age: 15 years; 52.9% males and 47.1% females), mostly diagnosed with neurological disorders, developmental and intellectual disabilities, behavioural and sensory disorders, cardiovascular and metabolic abnormalities, renal diseases and others. Screening identified 1,000 unique variants with heteroplasmy levels greater than 10% and 192 unique variants with 1-10% heteroplasmy, excluding hypervariable regions. Among these variants, four confirmed pathogenic variants were detected according to MITOMAP (m.1555 A > G, m.3243 A > G, m.9035T > C, and m.11778G > A), each identified in one patient. The application of pathogenicity and frequency criteria led to the identification of three unique variants and one in monozygotic twin sister with low levels of heteroplasmy, which were confirmed by next-generation sequencing. Finally, one of them, the variant m.15897G > A, was recognised as likely pathogenic (PP3, PS2). Our study highlights the complexity of diagnosing mitochondrial diseases associated with mtDNA mutations and emphasises the need for a comprehensive genotype-phenotype approach to correctly identify causal variants.
期刊介绍:
Neurogenetics publishes findings that contribute to a better understanding of the genetic basis of normal and abnormal function of the nervous system. Neurogenetic disorders are the main focus of the journal. Neurogenetics therefore includes findings in humans and other organisms that help understand neurological disease mechanisms and publishes papers from many different fields such as biophysics, cell biology, human genetics, neuroanatomy, neurochemistry, neurology, neuropathology, neurosurgery and psychiatry.
All papers submitted to Neurogenetics should be of sufficient immediate importance to justify urgent publication. They should present new scientific results. Data merely confirming previously published findings are not acceptable.