PTOV1与ZNF449相互作用促进结直肠癌的发展。

IF 5.2 1区 生物学 Q1 BIOLOGY
Zhiyong Wang, Xinwei Qiao, Kaming Xue, Qianzhi Chen, Anshu Li
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引用次数: 0

摘要

人们认为,PTOV1在包括前列腺癌在内的各种人类癌症中发挥着重要作用。然而,其在结直肠癌(CRC)中的临床意义和生物学作用尚不清楚。TCGA、NCBI/GEO和Kaplan-Meier图数据库挖掘为了解PTOV1在结直肠癌中的功能和临床意义提供了重要线索。Western blotting、免疫组织化学和免疫荧光检测CRC细胞系和组织中PTOV1蛋白水平。为了探索PTOV1在CRC发展中的作用及其潜在机制,进行了几项体外和体内研究,如CCK-8测定、集落形成、transwell测定、qRT-PCR、Co-IP、GST下拉、免疫染色和小鼠异种移植试验。结果表明,在人结直肠癌的组织和细胞中,PTOV1的表达水平上调。高表达的患者生存期较短。ZNF449在体外和体内与PTOV1相互作用并加速结直肠癌的发展。通过Co-IP和GST下拉研究,证明了PTOV1/ZNF449的物理相互作用。此外,PTOV1直接结合ZNF449,该复合体协同促进MYC的转录。此外,在异种移植小鼠模型中,TAT- PTOV1 (125-283 aa)蛋白破坏了PTOV1/ZNF449的相互作用,从而抑制了CRC的发展。根据这些发现,PTOV1在CRC的进展中起着重要作用,并且PTOV1/ZNF449相互作用可能是CRC的一个可能的治疗靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
PTOV1 interacts with ZNF449 to promote colorectal cancer development.

PTOV1 is recognized to have a significant role in various human cancers, including prostate cancer. However, it remains unclear what its clinical significance and biological role are in colorectal cancer (CRC). TCGA, NCBI/GEO, and Kaplan-Meier plot database mining provided important clues into the function and clinical importance of PTOV1 in CRC. Western blotting, immunohistochemistry, and immunofluorescence were utilized to discover PTOV1 protein levels in CRC cell lines and tissues. To explore the involvement of PTOV1 in the development of CRC and the underlying mechanisms, several in-vitro and in-vivo studies were executed, such as CCK-8 assays, colony formation, transwell assays, qRT-PCR, Co-IP, GST pull-down, immunostaining, and mouse xenograft assays. It was shown that PTOV1 expression level was upregulated in the tissues and cells of human CRC. PTOV1 high-expression level was associated with short survival. ZNF449 interacted with PTOV1 and accelerated CRC development in vitro and in vivo. Through Co-IP and GST pull-down studies, the physical interaction of PTOV1/ZNF449 was demonstrated. Furthermore, PTOV1 directly bound ZNF449, and this complex synergistically promoted the transcription of MYC. In addition, the PTOV1/ZNF449 interaction was disrupted by the TAT- PTOV1 (125-283 aa) protein leading to inhibit the CRC development in a xenografted mouse model. According to these findings, PTOV1 has an essential role in CRC progression, and PTOV1/ZNF449 interaction could be a possible therapeutic target for CRC.

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来源期刊
Communications Biology
Communications Biology Medicine-Medicine (miscellaneous)
CiteScore
8.60
自引率
1.70%
发文量
1233
审稿时长
13 weeks
期刊介绍: Communications Biology is an open access journal from Nature Research publishing high-quality research, reviews and commentary in all areas of the biological sciences. Research papers published by the journal represent significant advances bringing new biological insight to a specialized area of research.
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