黄酒致急性草酸肾病1例。

IF 2.2 4区 医学 Q2 UROLOGY & NEPHROLOGY
Xi Chen, Jie Shen, Yuxue Liu, Yingchun Ran, Mindong Chen, Rong Zhou
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引用次数: 0

摘要

背景:急性草酸肾病是一种罕见但未被充分认识的急性肾损伤(AKI)病因。高剂量维生素C、马马苋、花生引起继发性草酸肾病的报道有报道,但以饮用黄酒引起的病例很少。我们报告一位63岁男性因黄酒引起急性肾损伤。患者饮用黄酒500毫升/天,过去10年。超声引导下行肾活检,发现肾小管急性损伤,偏光显微镜下可见肾小管双折射结晶。建立24小时尿草酸排泄量,基线血清肌酐为407µmol/L。治疗3个月后,随后的实验室评估显示尿草酸排泄明显减少,血清肌酐正常。结论:草酸盐饮食摄入过量引起的急性继发性草酸肾病可能导致AKI。不明原因AKI患者的肾脏活检是重要的,在探讨AKI的原因时应注意饮食行为。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Acute oxalate nephropathy due to yellow rice wine: a case report.

Background: Acute oxalate nephropathy is a rare but potentially underrecognized cause of acute kidney injury (AKI). The reports of secondary oxalate nephropathy induced by high doses of vitamin C, portulaca oleracea, Peanut were described, but the cause as yellow wine consumption are rare. We report a 63-year-old man with acute kidney injury due to yellow rice wine. The patient has drunk yellow rice wine 500 mL/day for the past 10 years. He underwent an ultrasound-guided renal biopsy, which showed acute tubular injury and birefringent crystals were observed in the renal tubules under a polarizing microscope. The 24-hour urinary oxalate excretion established and the baseline serum creatinine of 407 µmol/L. Following 3 months therapy, subsequent laboratory evaluation demonstrated significant reduction in urinary oxalate excretion excretion and serum creatinine normal.

Conclusions: Acute secondary oxalate nephropathy due to excessive dietary intake of oxalate may lead to AKI. Kidney biopsies in unknown cause AKI patients is important and attention should be payed to food behaviors when reasons for AKI are explored.

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来源期刊
BMC Nephrology
BMC Nephrology UROLOGY & NEPHROLOGY-
CiteScore
4.30
自引率
0.00%
发文量
375
审稿时长
3-8 weeks
期刊介绍: BMC Nephrology is an open access journal publishing original peer-reviewed research articles in all aspects of the prevention, diagnosis and management of kidney and associated disorders, as well as related molecular genetics, pathophysiology, and epidemiology.
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