ING4缺失可增强多能祖细胞的造血再生。

IF 2.6 3区 综合性期刊 Q1 MULTIDISCIPLINARY SCIENCES
PLoS ONE Pub Date : 2025-02-14 eCollection Date: 2025-01-01 DOI:10.1371/journal.pone.0316256
Georgina A Anderson, Marco Hernandez, Carlos Alfaro Quinde, Zanshé Thompson, Vera Binder-Blaser, Alison M Taylor, Katie L Kathrein
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引用次数: 0

摘要

尽管它在生存中起着至关重要的作用,但造血的许多方面仍未得到解决。在造血程序的经典模型中,静态造血干细胞(hsc)位于造血层次的顶端,具有自我更新和根据需要分化的能力。造血干细胞产生更多的增殖祖细胞,这些祖细胞具有多能潜能,但大部分或完全丧失了自我更新能力。在这里,我们已经确定了肿瘤抑制因子,生长抑制因子4 (ING4),作为多能祖细胞(MPP)稳态的关键调节因子。在缺乏ING4的情况下,我们发现mpp表达造血激活的转录程序,但它们在低水平的活性氧下保持静止。在功能上,缺乏ing4的MPPs在竞争性骨髓移植后具有强大的再生能力,与野生型MPPs相比,其血液嵌合性显著提高。这些数据表明,ING4缺陷促进了mpp的平衡状态,静止但转录激活,能够在压力下将平衡状态转化为强大的重新种群。我们的模型为进一步识别和表征控制mpp静止和再生的途径提供了关键工具。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Loss of ING4 enhances hematopoietic regeneration in multipotent progenitor cells.

Loss of ING4 enhances hematopoietic regeneration in multipotent progenitor cells.

Loss of ING4 enhances hematopoietic regeneration in multipotent progenitor cells.

Loss of ING4 enhances hematopoietic regeneration in multipotent progenitor cells.

Despite its critical role in survival, many aspects of hematopoiesis remain unresolved. In the classical model of the hematopoietic program, quiescent hematopoietic stem cells (HSCs) sit at the top of the hematopoietic hierarchy, with the ability to self-renew and differentiate as needed. HSCs give rise to more proliferative progenitor cells, which possess multipotent potential, but have largely or completely lost self-renewal capabilities. Here, we have identified the tumor suppressor, Inhibitor of Growth 4 (ING4), as a critical regulator of multipotent progenitor (MPP) homeostasis. In the absence of ING4, we show that MPPs express a transcriptional program of hematopoietic activation, yet they remain quiescent with low levels of reactive oxygen species. Functionally, ING4-deficient MPPs are capable of robust regeneration following competitive bone marrow transplantation, resulting in substantially higher blood chimerism compared to wild-type MPPs. These data suggest ING4 deficiency promotes a poised state in MPPs, quiescent but transcriptionally primed for activation, and capable of converting the poised state into robust repopulation upon stress. Our model provides key tools for further identification and characterization of pathways that control quiescence and regeneration in MPPs.

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来源期刊
PLoS ONE
PLoS ONE 生物-生物学
CiteScore
6.20
自引率
5.40%
发文量
14242
审稿时长
3.7 months
期刊介绍: PLOS ONE is an international, peer-reviewed, open-access, online publication. PLOS ONE welcomes reports on primary research from any scientific discipline. It provides: * Open-access—freely accessible online, authors retain copyright * Fast publication times * Peer review by expert, practicing researchers * Post-publication tools to indicate quality and impact * Community-based dialogue on articles * Worldwide media coverage
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