{"title":"修正“在老年脑出血小鼠模型中靶向miR-124/铁转运蛋白信号通过抑制细胞凋亡和铁下垂改善神经元细胞死亡”","authors":"","doi":"10.1111/acel.70006","DOIUrl":null,"url":null,"abstract":"<p>Bao, W.-D., X.-T. Zhou, L.-T. Zhou, et al. 2020. “Targeting miR-124/Ferroportin Signaling Ameliorated Neuronal Cell Death Through Inhibiting Apoptosis and Ferroptosis in Aged Intracerebral Hemorrhage Murine Model.” <i>Aging Cell</i> 19: e13235. https://doi.org/10.1111/acel.13235.</p><p>During the data organization of this manuscript, there was an error inadvertently incorporated into the manuscript. We mistakenly placed a duplicated image of iron staining for two control groups: the ICH and con virus group in Figure 2E. All other parts of this article remain intact, valid, and unchanged.</p><p>The authors apologize for this error.</p>","PeriodicalId":55543,"journal":{"name":"Aging Cell","volume":"24 3","pages":""},"PeriodicalIF":7.8000,"publicationDate":"2025-02-13","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1111/acel.70006","citationCount":"0","resultStr":"{\"title\":\"Correction to “Targeting miR-124/ Ferroportin Signaling Ameliorated Neuronal Cell Death Through Inhibiting Apoptosis and Ferroptosis in Aged Intracerebral Hemorrhage Murine Model”\",\"authors\":\"\",\"doi\":\"10.1111/acel.70006\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Bao, W.-D., X.-T. Zhou, L.-T. Zhou, et al. 2020. “Targeting miR-124/Ferroportin Signaling Ameliorated Neuronal Cell Death Through Inhibiting Apoptosis and Ferroptosis in Aged Intracerebral Hemorrhage Murine Model.” <i>Aging Cell</i> 19: e13235. https://doi.org/10.1111/acel.13235.</p><p>During the data organization of this manuscript, there was an error inadvertently incorporated into the manuscript. We mistakenly placed a duplicated image of iron staining for two control groups: the ICH and con virus group in Figure 2E. All other parts of this article remain intact, valid, and unchanged.</p><p>The authors apologize for this error.</p>\",\"PeriodicalId\":55543,\"journal\":{\"name\":\"Aging Cell\",\"volume\":\"24 3\",\"pages\":\"\"},\"PeriodicalIF\":7.8000,\"publicationDate\":\"2025-02-13\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1111/acel.70006\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Aging Cell\",\"FirstCategoryId\":\"99\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/acel.70006\",\"RegionNum\":1,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"Biochemistry, Genetics and Molecular Biology\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Aging Cell","FirstCategoryId":"99","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/acel.70006","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"Biochemistry, Genetics and Molecular Biology","Score":null,"Total":0}
Correction to “Targeting miR-124/ Ferroportin Signaling Ameliorated Neuronal Cell Death Through Inhibiting Apoptosis and Ferroptosis in Aged Intracerebral Hemorrhage Murine Model”
Bao, W.-D., X.-T. Zhou, L.-T. Zhou, et al. 2020. “Targeting miR-124/Ferroportin Signaling Ameliorated Neuronal Cell Death Through Inhibiting Apoptosis and Ferroptosis in Aged Intracerebral Hemorrhage Murine Model.” Aging Cell 19: e13235. https://doi.org/10.1111/acel.13235.
During the data organization of this manuscript, there was an error inadvertently incorporated into the manuscript. We mistakenly placed a duplicated image of iron staining for two control groups: the ICH and con virus group in Figure 2E. All other parts of this article remain intact, valid, and unchanged.
期刊介绍:
Aging Cell, an Open Access journal, delves into fundamental aspects of aging biology. It comprehensively explores geroscience, emphasizing research on the mechanisms underlying the aging process and the connections between aging and age-related diseases.