杀虫剂喹磷基Kinalux 25EC对鲈鱼幼鱼的毒性:脑胆碱酯酶和生长抑制

Q1 Environmental Science
Nguyen Van Cong , Tran Hoang Kha , Bui Thi Bich Hang , Duong Tri Dung , Tran Sy Nam
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引用次数: 0

摘要

本研究旨在测定四磷对鲈鱼幼鱼脑胆碱酯酶(ChE)和生长的中位致死浓度(LC50)和亚致死水平的影响。结果表明,LC50-96 h为1.88 mg L−1。10% LC50-96h或更高浓度的脑ChE活性显著抑制,直至暴露96h结束。在生长试验中,10%和20% LC50-96h处理的特定生长率分别被抑制48.0%和34.8%,而10%和20% LC50-96h处理的饲料系数分别提高到164.5%和132.3%。10% LC50-96h存活率为86%,20% LC50-96h存活率为34.7%。该研究表明,喹磷磷对生活在湄公河三角洲下游稻田的攀缘鲈具有剧毒,对攀缘鲈有潜在的健康风险。监测和控制水生环境中五磷的含量是减轻其对水生生物不利影响的重要举措。迫切需要推广使用毒性较小或可生物降解的农药。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Toxicity of insecticide quinalphos-based Kinalux 25EC on fingerling climbing perch (Anabas testudineus): Brain cholinesterase and growth inhibitions
This study aims to determine median lethal concentration (LC50) and effects at sublethal levels of quinalphos on brain cholinesterase (ChE) and growth of fingerlings climbing perch (Anabas testudineus). Results showed that LC50-96 h was 1.88 mg L−1. Brain ChE activity was significantly inhibited at concentration of 10 % LC50-96h or higher up to the end 96 h of exposure. In the growth experiment, specific growth rate was inhibited 48.0 % and 34.8 % at treatment 10 % and 20 % LC50-96h, respectively while feed conversion ratio elevated to 164.5 % control at treatment 10 % and to 132.3 % at treatment 20 % LC50-96h. Survival rates were 86 % at 10 % LC50-96h and 34.7 % at 20 % LC50-96h. The study indicated that quinalphos is highly toxic for the climbing perch leading to potential health risk for this species living in rice fields of the Lower Mekong Delta. Monitoring and controlling quinalphos levels in aquatic environments are important acts to mitigating its adverse impacts on aquatic organisms. Promoting the use of less toxic or biodegradable pesticides is urgently needed.
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来源期刊
Case Studies in Chemical and Environmental Engineering
Case Studies in Chemical and Environmental Engineering Engineering-Engineering (miscellaneous)
CiteScore
9.20
自引率
0.00%
发文量
103
审稿时长
40 days
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