小鼠妊娠期高脂肪饮食损害胎儿心脏发育。

IF 1.2 4区 医学 Q3 CARDIAC & CARDIOVASCULAR SYSTEMS
Kaixin Ge, Diyaerjiang Aierken, Defang Deng, Zhen Jiang, Teng Feng, Jufeng Meng, Hui Zhang, Jinjun Cao, Chen Liu
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引用次数: 0

摘要

母亲营养过剩与后代的有害后果有关。然而,妊娠期暴露于高脂肪饮食(HFD)对胎儿发育的具体影响尚不清楚。本研究旨在阐明暴露于妊娠期HFD的小鼠的新生器官和心肌细胞的发育表型,揭示生长迟缓和心肌细胞细胞周期活性显著降低。本研究采用HFD模型研究母体HFD对子代发育的影响。在后代中观察到发育缺陷,并且由于母体HFD,在新生儿器官中注意到严重的细胞增殖限制。基于这一证据,我们发现暴露于母体HFD的后代心肌细胞增殖减少。此外,RNA测序分析显示,HFD降低了出生后0天(P0)心肌细胞的脂肪酸代谢,增强了炎症反应,并上调了参与tp53调控的细胞周期停滞的基因的转录。此外,我们的研究结果表明,怀孕期间母亲饮食的影响是深远的,正常的哺乳和分娩后喂养不能帮助成年后代从心脏发育缺陷中恢复过来。这些发现强调了受母体HFD影响的多种途径,特别是暗示了一种潜在的tp53依赖机制,有助于后代的心脏缺陷。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
High-Fat Diet during Mouse Pregnancy Impairs Fetal Heart Development.

Maternal overnutrition correlates with detrimental outcomes in offspring. However, the specific effects of gestational exposure to a high-fat diet (HFD) on fetal development remain unclear. This study aimed to elucidate the developmental phenotypes of neonatal organs and cardiomyocytes of mice exposed to gestational HFD, revealing growth retardation and a notable reduction in cardiomyocyte cell cycle activity. In this study, an HFD model was used to investigate the effects of maternal HFD on offspring development. Defective development was observed in the offspring, and severe restriction of cell proliferation was noted in the neonatal organs as a result of maternal HFD. Based on this evidence, we detected a reduction in cardiomyocyte proliferation in offspring exposed to maternal HFD. Moreover, RNA sequencing analysis revealed that HFD diminished fatty acid metabolism, enhanced the inflammatory response, and upregulated the transcription of genes involved in Tp53-regulated cell cycle arrest in postnatal day 0 (P0) cardiomyocytes. Furthermore, our results showed that the effects of the maternal diet during gestation are profound and normal lactation and feeding after delivery cannot help adult offspring recover from defective heart development. These findings highlight the diverse pathways affected by maternal HFD, particularly implicating a potential TP53-dependent mechanism contributing to cardiac defects in offspring.

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来源期刊
International heart journal
International heart journal 医学-心血管系统
CiteScore
2.50
自引率
6.70%
发文量
148
审稿时长
6-12 weeks
期刊介绍: Authors of research articles should disclose at the time of submission any financial arrangement they may have with a company whose product figures prominently in the submitted manuscript or with a company making a competing product. Such information will be held in confidence while the paper is under review and will not influence the editorial decision, but if the article is accepted for publication, the editors will usually discuss with the authors the manner in which such information is to be communicated to the reader.
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