Sarah J Hammond, Andrea J Roff, Joshua L Robinson, Jack R T Darby, Ashley S Meakin, Vicki L Clifton, Robert J Bischof, Michael J Stark, Megan J Wallace, Andrew Tai, Janna L Morrison, Kathryn L Gatford
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引用次数: 0
摘要
母体哮喘(MA)在子宫内的暴露与儿童期随后的哮喘风险之间的关联机制尚不完全清楚。有报道称,婴儿和儿童的气道会发生病理性重塑,包括网状基底膜增厚,并在儿童期发展为哮喘。这表明,出生前气道发育的改变是子宫内暴露于 MA 的儿童患哮喘风险增加的潜在机制。我们假设,子宫内暴露于 MA 会使新生儿的气道直径减小,气道相关平滑肌面积和网状基底膜厚度增加。实验性 MA 是通过母体致敏诱导的,然后在怀孕前和怀孕期间用屋尘螨挑战气道。对照组(n = 16)或 MA 组(n = 26)母羊的羔羊在妊娠 140 天(足月 = 150 天)时分娩,通气 45 分钟,然后人道处死。左肺充气固定,收集第 2-5 代气道的横切面。气道切片用血苏木精和伊红、马森三色染色法以及戈登和斯威特组织学染色法进行形态学分析。各组羔羊的体重和肺重量相似(P > 0.5 和 P > 0.7)。与未接触 MA 的对照组羔羊相比,接触 MA 的羔羊气道直径更窄(P = 0.019),网状基底膜更薄(P = 0.016),但气道相关平滑肌面积相似(P = 0.152)。我们的研究结果表明,母亲患有哮喘的羔羊罹患哮喘的风险增加的潜在机制与遗传风险或孕期行为变化无关。
In utero exposure to experimental maternal asthma alters fetal airway development in sheep.
The mechanisms linking maternal asthma (MA) exposure in utero and subsequent risk of asthma in childhood are not fully understood. Pathological airway remodelling, including reticular basement membrane thickening, has been reported in infants and children who go on to develop asthma later in childhood. This suggests altered airway development before birth as a mechanism underlying increased risk of asthma in children exposed in utero to MA. We hypothesised that in utero MA exposure would reduce airway diameter and increase airway-associated smooth muscle area and reticular basement membrane thickness in neonatal offspring. Experimental MA was induced by maternal sensitisation followed by airway challenges with house dust mite before and during pregnancy. Lambs from control (n = 16) or MA (n = 26) ewes were delivered at ∼140 days gestation (term = 150 days), ventilated for 45 min, then humanely killed. Left lungs were inflation-fixed, and cross-sections of generation 2-5 airways were collected. Airway sections were stained with Haematoxylin and Eosin, Masson's Trichrome and Gordon and Sweet's histological stains for morphological analysis. Lamb body and lung weights were similar between groups (P > 0.5 and P > 0.7, respectively). Lambs that were exposed to MA had narrower airway diameters (P = 0.019) and thinner reticular basement membrane (P = 0.016) but similar airway-associated smooth muscle area (P = 0.152) compared with unexposed control lambs. Our results demonstrate a potential mechanism for increased risk of asthma in children of mothers with asthma, independent of genetic risk or behavioural changes during pregnancy.
期刊介绍:
Experimental Physiology publishes research papers that report novel insights into homeostatic and adaptive responses in health, as well as those that further our understanding of pathophysiological mechanisms in disease. We encourage papers that embrace the journal’s orientation of translation and integration, including studies of the adaptive responses to exercise, acute and chronic environmental stressors, growth and aging, and diseases where integrative homeostatic mechanisms play a key role in the response to and evolution of the disease process. Examples of such diseases include hypertension, heart failure, hypoxic lung disease, endocrine and neurological disorders. We are also keen to publish research that has a translational aspect or clinical application. Comparative physiology work that can be applied to aid the understanding human physiology is also encouraged.
Manuscripts that report the use of bioinformatic, genomic, molecular, proteomic and cellular techniques to provide novel insights into integrative physiological and pathophysiological mechanisms are welcomed.