在少突胶质细胞命运决定过程中procaspase-3的短暂上调。

IF 4.4 2区 医学 Q1 NEUROSCIENCES
Yasmine Kamen, Timothy W Chapman, Enrique T Piedra, Matthew E Ciolkowski, Robert A Hill
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引用次数: 0

摘要

少突胶质细胞是在整个生命过程中以及神经退行性疾病中由脑内少突胶质前体细胞(OPCs)产生的。从OPC到少突胶质细胞的转变涉及复杂的分子级联和形态学状态,这些状态使细胞做出命运决定,是整合为髓鞘少突胶质细胞还是通过凋亡死亡。少突胶质细胞成熟影响退行性疾病中发生的细胞死亡机制,但是当OPCs转变为少突胶质细胞时,细胞死亡机制是否以及如何改变尚不清楚。在这里,我们发现分化的少突胶质细胞在雌性和雄性小鼠中都会短暂上调酶原procaspase-3,使这些细胞在分化过程中做出生存决定。药物抑制caspase-3可降低少突胶质细胞密度,表明procaspase-3上调与少突胶质细胞的成功生成有关。此外,使用procaspase-3作为标记,我们发现少突胶质细胞分化在老化的皮层和白质中继续存在。综上所述,我们的数据确定了procaspase-3作为分化少突胶质细胞的标记物,并提供了在决定整合或死亡过程中发生的潜在机制的见解。髓磷脂功能障碍和少突胶质细胞死亡发生在几种神经系统疾病和衰老中。虽然在整个生命周期和神经退行性疾病中,新的少突胶质细胞可以由少突胶质细胞前体细胞产生,但这一过程效率低下,许多分化的少突胶质细胞无法整合并死亡。由于缺乏精确描述分化阶段的分子标记,研究调节这些命运决定的细胞检查点变得复杂。在这里,我们发现少突胶质细胞在分化过程中短暂上调酶原procaspase-3,建立procaspase-3作为分化少突胶质细胞的标记物。重要的是,procaspase-3的上调使分化的少突胶质细胞在死亡或整合之间做出命运决定,并促进少突胶质细胞分化。因此,我们的数据揭示了指导少突胶质细胞命运决定机制的新见解。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Transient Upregulation of Procaspase-3 during Oligodendrocyte Fate Decisions.

Oligodendrocytes are generated throughout life and in neurodegenerative conditions from brain resident oligodendrocyte precursor cells (OPCs). The transition from OPC to oligodendrocyte involves a complex cascade of molecular and morphological states that position the cell to make a fate decision to integrate as a myelinating oligodendrocyte or die through apoptosis. Oligodendrocyte maturation impacts the cell death mechanisms that occur in degenerative conditions, but it is unclear if and how the cell death machinery changes as OPCs transition into oligodendrocytes. Here, we discovered that differentiating oligodendrocytes transiently upregulate the zymogen procaspase-3 in both female and male mice, equipping these cells to make a survival decision during differentiation. Pharmacological inhibition of caspase-3 decreases oligodendrocyte density, indicating that procaspase-3 upregulation is linked to successful oligodendrocyte generation. Moreover, using procaspase-3 as a marker, we show that oligodendrocyte differentiation continues in the aging cortex and white matter. Taken together, our data establish procaspase-3 as a differentiating oligodendrocyte marker and provide insight into the underlying mechanisms occurring during the decision to integrate or die.

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来源期刊
Journal of Neuroscience
Journal of Neuroscience 医学-神经科学
CiteScore
9.30
自引率
3.80%
发文量
1164
审稿时长
12 months
期刊介绍: JNeurosci (ISSN 0270-6474) is an official journal of the Society for Neuroscience. It is published weekly by the Society, fifty weeks a year, one volume a year. JNeurosci publishes papers on a broad range of topics of general interest to those working on the nervous system. Authors now have an Open Choice option for their published articles
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