总结：整合RNA表达和选择性多聚腺苷化分析鉴定了肺腺癌中cps1 - ccdc137致癌轴。

IF 4.4 3区医学 Q2 ENVIRONMENTAL SCIENCES

Environmental Toxicology Pub Date : 2025-01-22 DOI:10.1002/tox.24478

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引用次数: 0

摘要

引用本文：徐东，李恒，李本超，李文杰，李高峰，“cps1 - ccdc137基因在肺腺癌中的表达及表达分析”，《环境毒理学杂志》，第39期。4 (2024): 2405-2416, https://doi.org/10.1002/tox.24105。上述文章于2024年1月4日在线发表在Wiley在线图书馆（http://onlinelibrary.wiley.com/）上，经期刊主编Paul B. Tchounwou；和Wiley期刊有限责任公司。经过出版商的调查，双方得出结论，这篇文章完全是在妥协的同行评议过程的基础上被接受的。因此，这篇文章必须撤回。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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RETRACTION: Integrated RNA Expression and Alternative Polyadenylation Analysis Identified CPSF1-CCDC137 Oncogenic Axis in Lung Adenocarcinoma.

Retraction: X. Xudong, L. Heng, C. Benchao, C. Wenjie, L. Bao, and L. Gaofeng, "Integrated RNA Expression and Alternative Polyadenylation Analysis Identified CPSF1-CCDC137 Oncogenic Axis in Lung Adenocarcinoma," Environmental Toxicology 39, no. 4 (2024): 2405-2416, https://doi.org/10.1002/tox.24105. The above article, published online on 4 January 2024, in Wiley Online Library (http://onlinelibrary.wiley.com/), has been retracted by agreement between the journal Editor-in-Chief, Paul B. Tchounwou; and Wiley Periodicals LLC. Following an investigation by the publisher, the parties have concluded that this article was accepted solely on the basis of a compromised peer review process. Therefore, the article must be retracted.

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来源期刊

Environmental Toxicology 环境科学-毒理学

CiteScore

7.10

自引率

8.90%

发文量

261

审稿时长

4.5 months

期刊介绍： The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.