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IF 25.5 1区医学 Q1 IMMUNOLOGY

Immunity Pub Date : 2025-01-14 DOI:10.1016/j.immuni.2024.12.007

Shuangshuang Yang, Guannan Huang, Jenny P.-Y. Ting

引用次数: 0

摘要

线粒体在内在凋亡和NLRP3炎症小体激活中发挥着关键作用，但这些过程如何相互关联仍不清楚。在本期《免疫》杂志上，Saller 等人揭示了 NLRP3 激活因子的复杂性，强调了线粒体在将细胞凋亡转换为 NLRP3 炎症小体激活过程中的作用。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

查看原文本刊更多论文

Mitochondria and NLRP3: To die or inflame

Mitochondria play critical roles in intrinsic apoptosis and NLRP3 inflammasome activation, but how these processes are interconnected remains unclear. In this issue of Immunity, Saller et al. unveiled the complexity of NLRP3 activators, highlighting mitochondria’s roles in switching apoptosis to NLRP3 inflammasome activation.

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来源期刊

Immunity 医学-免疫学

CiteScore

49.40

自引率

2.20%

发文量

205

审稿时长

6 months

期刊介绍： Immunity is a publication that focuses on publishing significant advancements in research related to immunology. We encourage the submission of studies that offer groundbreaking immunological discoveries, whether at the molecular, cellular, or whole organism level. Topics of interest encompass a wide range, such as cancer, infectious diseases, neuroimmunology, autoimmune diseases, allergies, mucosal immunity, metabolic diseases, and homeostasis.