回归：长非编码 RNA LINC00958 通过与宫颈癌中的 microRNA-5095 结合，通过 RRM2 调节细胞对放疗的敏感性。

IF 4.5 2区生物学 Q2 CELL BIOLOGY

Journal of Cellular Physiology Pub Date : 2025-01-13 DOI:10.1002/jcp.31525

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引用次数: 0

摘要

撤回：赵宏，g.h - h。郑,G.-C。李立新，杨永生。Wang， Y. Chen，和x - m。郑，“长链非编码RNA LINC00958通过RRM2结合microRNA-5095调节宫颈癌细胞对放疗的敏感性”，细胞生理学杂志，第234期。12(2019): 23349-23359。https://doi.org/10.1002/jcp.28902。上述文章于2019年6月6日在线发表在Wiley在线图书馆（wileyonlinelibrary.com）上，经期刊主编Robert Heath同意撤回；第三方报告在本文的图6a和另一篇文章的图7B之间检测到相同的肿瘤图像（Guo, et al. 2019 [https://doi.org/10.18632/aging.102271]]）。第三方报告了本文中的图2B与不同作者的其他文章之间的额外图像重复(Guo, et al. 2018 [https://doi.org/10.1186/s12964-018-0290-6]；刘，等。2019 [https://doi.org/10.1002/jcp.27893]；文，等。2019 [https://doi.org/10.1096/fj.201900310 R]；欧等。2020 [https://doi.org/10.1016/j.ebiom.2020.102694].]每篇提到的文章都描述了不同的科学背景。作者没有回应出版商的询问。由于与其他文章的图像重复的证据从根本上损害了本文报告的结论，因此已同意撤回。作者没有回应我们关于撤稿的通知。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

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RETRACTION: Long Noncoding RNA LINC00958 Regulates Cell Sensitivity to Radiotherapy Through RRM2 by Binding to microRNA-5095 in Cervical Cancer

RETRACTION: H. Zhao, G.-H. Zheng, G.-C. Li, L. Xin, Y.-S. Wang, Y. Chen, and X.-M. Zheng, “Long Noncoding RNA LINC00958 Regulates Cell Sensitivity to Radiotherapy Through RRM2 by Binding to microRNA-5095 in Cervical Cancer,” Journal of Cellular Physiology 234, no. 12 (2019): 23349-23359. https://doi.org/10.1002/jcp.28902.

The above article, published online on 06 June 2019 in Wiley Online Library (wileyonlinelibrary.com), and has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. A third party reported that the same tumor image was detected between Figure 6 A in this article and Figure 7B in another article (Guo, et al. 2019 [https://doi.org/10.18632/aging.102271]. The third party reported additional image duplications between Figure 2B in this article and other articles by different authors (Guo, et al. 2018 [https://doi.org/10.1186/s12964-018-0290-6]; (Liu, et al. 2019 [https://doi.org/10.1002/jcp.27893]; (Wen, et al. 2019 [https://doi.org/10.1096/fj.201900310 R]; and (Ou, et al. 2020 [https://doi.org/10.1016/j.ebiom.2020.102694]. Each mentioned article describes a different scientific context. The authors did not respond to an inquiry by the publisher. The retraction has been agreed on because the evidence of image duplication with other articles fundamentally compromises the conclusions reported in this article. The authors did not respond to our notice regarding the retraction.

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来源期刊

Journal of Cellular Physiology 生物-生理学

CiteScore

14.70

自引率

0.00%

发文量

256

审稿时长

1 months

期刊介绍： The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.