{"title":"更正“小胶质褪黑素受体1通过激活lc3相关的体外吞噬作用降解病理性α -突触核蛋白”。","authors":"","doi":"10.1111/cns.70219","DOIUrl":null,"url":null,"abstract":"<p>Yao XY, Cao BE, Liu JY, Lv QK, Zhang JR, Cheng XY, Mao CJ, Ma QH, Wang F, Liu CF. “Microglial Melatonin Receptor 1 Degrades Pathological Alpha-Synuclein Through Activating LC3-Associated Phagocytosis In Vitro”. <i>CNS Neurosci Ther</i>. 2024;30(10):e70088. doi: 10.1111/cns.70088.</p><p>The authors would like to correct Figure 2B, as errors were made during its preparation for publication. We declare that these corrections do not alter the results or conclusions of this paper. The corrected version of Figure 2, including Figure 2B, is provided here.</p>","PeriodicalId":154,"journal":{"name":"CNS Neuroscience & Therapeutics","volume":"31 1","pages":""},"PeriodicalIF":4.8000,"publicationDate":"2025-01-13","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11726115/pdf/","citationCount":"0","resultStr":"{\"title\":\"Correction to “Microglial Melatonin Receptor 1 Degrades Pathological Alpha-Synuclein Through Activating LC3-Associated Phagocytosis In Vitro”\",\"authors\":\"\",\"doi\":\"10.1111/cns.70219\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>Yao XY, Cao BE, Liu JY, Lv QK, Zhang JR, Cheng XY, Mao CJ, Ma QH, Wang F, Liu CF. “Microglial Melatonin Receptor 1 Degrades Pathological Alpha-Synuclein Through Activating LC3-Associated Phagocytosis In Vitro”. <i>CNS Neurosci Ther</i>. 2024;30(10):e70088. doi: 10.1111/cns.70088.</p><p>The authors would like to correct Figure 2B, as errors were made during its preparation for publication. We declare that these corrections do not alter the results or conclusions of this paper. The corrected version of Figure 2, including Figure 2B, is provided here.</p>\",\"PeriodicalId\":154,\"journal\":{\"name\":\"CNS Neuroscience & Therapeutics\",\"volume\":\"31 1\",\"pages\":\"\"},\"PeriodicalIF\":4.8000,\"publicationDate\":\"2025-01-13\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11726115/pdf/\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"CNS Neuroscience & Therapeutics\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1111/cns.70219\",\"RegionNum\":1,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"NEUROSCIENCES\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"CNS Neuroscience & Therapeutics","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1111/cns.70219","RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"NEUROSCIENCES","Score":null,"Total":0}
Correction to “Microglial Melatonin Receptor 1 Degrades Pathological Alpha-Synuclein Through Activating LC3-Associated Phagocytosis In Vitro”
Yao XY, Cao BE, Liu JY, Lv QK, Zhang JR, Cheng XY, Mao CJ, Ma QH, Wang F, Liu CF. “Microglial Melatonin Receptor 1 Degrades Pathological Alpha-Synuclein Through Activating LC3-Associated Phagocytosis In Vitro”. CNS Neurosci Ther. 2024;30(10):e70088. doi: 10.1111/cns.70088.
The authors would like to correct Figure 2B, as errors were made during its preparation for publication. We declare that these corrections do not alter the results or conclusions of this paper. The corrected version of Figure 2, including Figure 2B, is provided here.
期刊介绍:
CNS Neuroscience & Therapeutics provides a medium for rapid publication of original clinical, experimental, and translational research papers, timely reviews and reports of novel findings of therapeutic relevance to the central nervous system, as well as papers related to clinical pharmacology, drug development and novel methodologies for drug evaluation. The journal focuses on neurological and psychiatric diseases such as stroke, Parkinson’s disease, Alzheimer’s disease, depression, schizophrenia, epilepsy, and drug abuse.