在kmt2a重排的儿童t细胞急性淋巴细胞白血病细胞中,一种独特的替代mRNA剪接谱鉴定了致癌CD44转录物变体3

IF 2.5 4区 医学 Q2 HEMATOLOGY
Amanda Ramilo Amor, Sabina Enlund, Indranil Sinha, Qingfei Jiang, Ola Hermanson, Anna Nilsson, Shahrzad Shirazi Fard, Frida Holm
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引用次数: 0

摘要

t细胞急性淋巴细胞白血病(T-ALL)占所有儿科all病例的10-15%,由于普遍存在遗传和染色体异常,其病理复杂而闻名。虽然大多数儿童都能成功治愈,但涉及KMT2A基因的染色体重排被认为是预后不良的因素。在171个儿童T-ALL样本队列中,我们研究了KMT2A重排(KMT2A-r) T-ALL与KMT2A阴性(KMT2A-wt) T-ALL样本在基因和剪接变异模式上的差异。我们的研究结果已经确定了儿童KMT2A-r患者样本中不同的基因表达和剪接变异表达模式,包括剪接调节标记ESRP1和MBNL3的显著表达。此外,与KMT2A-wt T-ALL样本相比,KMT2A-r中BCL2的促存活长转录变体上调。最后,在KMT2A-r T-ALL中,CD44启动子区激活甲基化水平升高,随后致癌转录物变体CD44v3上调。总之,这表明CD44v3可能在KMT2A-r重排T-ALL的基于基因表达的风险分层中发挥潜在作用,并可能作为使用剪接调节剂的治疗靶点。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
A distinct alternative mRNA splicing profile identifies the oncogenic CD44 transcript variant 3 in KMT2A-rearranged pediatric T-cell acute lymphoblastic leukemia cells.

T-cell acute lymphoblastic leukemia (T-ALL), which constitutes of 10-15% of all pediatric acute lymphoblastic leukemia (ALL) cases, is known for its complex pathology due to pervasive genetic and chromosomal abnormalities. Although most children are successfully cured, chromosomal rearrangements involving the KMT2A gene is considered a poor prognostic factor. In a cohort of 171 pediatric T-ALL samples, we have studied differences in gene and splice variant patterns in KMT2A-rearranged (KMT2A-r) T-ALL compared with KMT2A-negative (KMT2A-wt) T-ALL samples. Our results have identified a distinct gene expression and splice variant expression pattern in pediatric KMT2A-r patient samples including significant expression of splicing regulatory markers ESRP1 and MBNL3. Additionally, the prosurvival long transcript variant of BCL2 were upregulated in KMT2A-r compared with KMT2A-wt T-ALL samples. Lastly, increased levels of activating methylation in the promoter region of CD44 were identified followed by an upregulation of the oncogenic transcript variant CD44v3 in KMT2A-r T-ALL. Together, this suggests that CD44v3 could play a potential role as gene expression-based risk stratification of KMT2A-r T-ALL and could possibly serve as a therapeutic target using splicing modulators.

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来源期刊
Experimental hematology
Experimental hematology 医学-血液学
CiteScore
5.30
自引率
0.00%
发文量
84
审稿时长
58 days
期刊介绍: Experimental Hematology publishes new findings, methodologies, reviews and perspectives in all areas of hematology and immune cell formation on a monthly basis that may include Special Issues on particular topics of current interest. The overall goal is to report new insights into how normal blood cells are produced, how their production is normally regulated, mechanisms that contribute to hematological diseases and new approaches to their treatment. Specific topics may include relevant developmental and aging processes, stem cell biology, analyses of intrinsic and extrinsic regulatory mechanisms, in vitro behavior of primary cells, clonal tracking, molecular and omics analyses, metabolism, epigenetics, bioengineering approaches, studies in model organisms, novel clinical observations, transplantation biology and new therapeutic avenues.
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