撤回：冬凌草苷对支气管肺发育不良动物模型的影响。

IF 3 3区生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY

Journal of cellular biochemistry Pub Date : 2024-12-30 DOI:10.1002/jcb.30696

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引用次数: 0

摘要

引用本文：张淑娟，王军，辛志强，孙超，鞠志强，薛旭，姜文，辛琪，王军，张志强，栾毅，“冬凌草苷对支气管肺发育不良动物模型的影响”，《细胞生物化学》，第125期。9 (2024): e30632, https://doi.org/10.1002/jcb.30632。上述文章于2024年7月16日在Wiley在线图书馆（wileyonlinelibrary.com）上发表，经该杂志主编Christian Behl；和Wiley期刊有限责任公司。在发表之后，该杂志注意到，这篇文章完全是基于折衷的同行评议过程而被接受的。此外，多重结论没有得到充分的数据支持。因此，这篇文章被撤回，因为编辑认为其结论无效。

本文章由计算机程序翻译，如有差异，请以英文原文为准。

查看原文本刊更多论文

RETRACTION: Effect of Oridonin on Experimental Animal Model of Bronchopulmonary Dysplasia

RETRACTION: S. Zhang, J. Wang, Z. Xin, C. Sun, Z. Ju, X. Xue, W. Jiang, Q. Xin, J. Wang, Z. Zhang, and Y. Luan, “Effect of Oridonin on Experimental Animal Model of Bronchopulmonary Dysplasia,” Journal of Cellular Biochemistry 125, no. 9 (2024): e30632, https://doi.org/10.1002/jcb.30632.

The above article, published online on 16 July 2024 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. Following publication, it has come to the attention of the journal that the article was accepted solely on the basis of compromised peer review processes. Furthermore, multiple conclusions are not sufficiently supported by the data. Accordingly, the article is retracted as the editors consider its conclusions to be invalid.

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来源期刊

Journal of cellular biochemistry 生物-生化与分子生物学

CiteScore

9.90

自引率

0.00%

发文量

164

审稿时长

1 months

期刊介绍： The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.