左外侧通道AVRT引起窦房结过度抑制和可逆性心肌病1例报告。

Q3 Medicine
Avinash Jeewooth , Atul Kaushik , Aparna Jaswal , Vikas Kumar
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引用次数: 0

摘要

通过房窦入口阻断,窦房结免受其他组织的过度抑制或快速心律失常的影响。一位57岁的女性表现为症状性心动过速综合征和严重的左室功能障碍。电生理研究显示存在左外侧副通路。消融副通路后,不再能诱发心动过速。她的左室收缩功能在随访中明显改善,外循环记录仪显示无心动过缓发作。因此,我们认为心动过缓是由AVRT引起的窦房结过度抑制引起的,可逆性心肌病也是由高心动过速负担引起的。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Left lateral pathway AVRT causing overdrive suppression of SA node and reversible cardiomyopathy: A case report
The SA node is protected from the overdrive suppression by the other tissues or tachyarrhythmias by means of atrio-sinus entrance block. A 57-year-old woman presented with symptomatic tachy-brady syndrome and severe LV dysfunction. Electrophysiological study revealed presence of left lateral accessory pathway. After ablation of the accessory pathway, tachycardia could no longer be induced. Her LV systolic function significantly improved on follow up and an external loop recorder showed no episodes of bradycardia. Therefore, we conclude that the bradycardia was due to overdrive suppression of SA node caused by AVRT, and the reversible cardiomyopathy was also due to high tachycardia burden.
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来源期刊
Indian Pacing and Electrophysiology Journal
Indian Pacing and Electrophysiology Journal Medicine-Cardiology and Cardiovascular Medicine
CiteScore
2.20
自引率
0.00%
发文量
91
审稿时长
61 days
期刊介绍: Indian Pacing and Electrophysiology Journal is a peer reviewed online journal devoted to cardiac pacing and electrophysiology. Editorial Advisory Board includes eminent personalities in the field of cardiac pacing and electrophysiology from Asia, Australia, Europe and North America.
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