在膀胱癌中,LncRNA SnoRNA宿主基因6的上调通过竞争性结合MicroRNA-125b并与Snail1/2相互作用调控NUAK家族snf1样激酶-1的表达。

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

摘要

引用本文:王超,陶伟,倪淑娟,陈清,“通过MicroRNA-125b的竞争结合和Snail1/2的相互作用,LncRNA - SnoRNA宿主基因6的上调调控NUAK家族snf1样激酶-1的表达”,《细胞生物化学》第12期,第1期。1 (2019): 357-367, https://doi.org/10.1002/jcb.27387。上述文章于2018年8月30日在线发表在Wiley在线图书馆(wileyonlinelibrary.com)上,经作者同意撤回;杂志主编克里斯蒂安·贝尔;和Wiley期刊有限责任公司。由于第三方对文章中提供的数据提出了担忧,已经同意撤回。具体来说,图1c、2c、2e、3a、3b和3c中的多个图像元素被发现在其他地方和不同的科学背景下发表过。因此,编者认为本文的结论无效。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Upregulation of LncRNA SnoRNA Host Gene 6 Regulates NUAK Family SnF1-like Kinase-1 Expression by Competitively Binding MicroRNA-125b and Interacting with Snail1/2 in Bladder Cancer

RETRACTION: C. Wang, W. Tao, S. Ni, Q. Chen, “Upregulation of LncRNA SnoRNA Host Gene 6 Regulates NUAK Family SnF1-like Kinase-1 Expression by Competitively Binding MicroRNA-125b and Interacting with Snail1/2 in Bladder Cancer,” Journal of Cellular Biochemistry 120, no. 1 (2019): 357-367, https://doi.org/10.1002/jcb.27387.

The above article, published online on 30 August 2018 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the authors; the journal Editor-in-Chief, Christian Behl; and Wiley Periodicals LLC. The retraction has been agreed to concerns raised by third parties on the data presented in the article. Specifically, multiple image elements in Figures 1c, 2c, 2e, 3a, 3b, and 3c were found to have been published elsewhere and in a different scientific context. Accordingly, the editors consider the conclusions of this article to be invalid.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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