回放:苯乙酰谷氨酰胺(PAGln)通过β1肾上腺素能受体增强心肌梗死后心肌细胞的死亡。

IF 4.4 3区 医学 Q2 ENVIRONMENTAL SCIENCES
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引用次数: 0

摘要

撤回:H. Xu, N. Yang, B.Y. Wang, L. Zhou, L.L. Xu, Y. Chen, D.J. Wang, and W.H. Ge, "Phenylacetyl Glutamine (PAGln) Enhances Cardiomyocyte Death after Myocardial Infarction through β1 Adrengic Receptor," Environmental Toxicology 39, no.3 (2024):1682-1699, https://doi.org/10.1002/tox.24063.上述文章于 2023 年 12 月 02 日在线发表于 Wiley Online Library (http://onlinelibrary.wiley.com/),经期刊主编 Paul B. Tchounwou 和 Wiley Periodicals LLC 协议,该文章已被撤回。经过出版商的调查,双方得出结论认为,这篇文章被接受的唯一依据是同行评审程序有问题。此外,调查发现这篇文章与之前发表的一篇文章(Beale et al.)因此,这篇文章必须撤回。作者葛伟宏和徐航不同意这一决定。其他作者未作回应。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Phenylacetyl Glutamine (PAGln) Enhances Cardiomyocyte Death After Myocardial Infarction Through β1 Adrenergic Receptor.

Retraction: H. Xu, N. Yang, B.Y. Wang, L. Zhou, L.L. Xu, Y. Chen, D.J. Wang, and W.H. Ge, "Phenylacetyl Glutamine (PAGln) Enhances Cardiomyocyte Death after Myocardial Infarction through β1 Adrenergic Receptor," Environmental Toxicology 39, no. 3 (2024): 1682-1699, https://doi.org/10.1002/tox.24063. The above article, published online on 02 December 2023, in Wiley Online Library (http://onlinelibrary.wiley.com/), has been retracted by agreement between the journal Editor-in-Chief, Paul B. Tchounwou; and Wiley Periodicals LLC. Following an investigation by the publisher, the parties have concluded that this article was accepted solely on the basis of a compromised peer review process. In addition, the investigation found there was significant unattributed textual overlap between this article and a previously-published article (Beale et al., 2008; https://doi.org/10.1186/1471-2407-8-200). Therefore, the article must be retracted. The authors Weihong Ge and Hang Xu disagree with this decision. The other authors did not respond.

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来源期刊
Environmental Toxicology
Environmental Toxicology 环境科学-毒理学
CiteScore
7.10
自引率
8.90%
发文量
261
审稿时长
4.5 months
期刊介绍: The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are: Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration; Natural toxins and their impacts; Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation; Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard; Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.
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