通过调节 miR-143-3P/MAPK/MCP-1、ERK5/CXCR4、Orai-1/STIM-1 信号通路,联合地奥司明和比索洛尔减轻氯化钴诱导的心脏毒性和内皮功能障碍》[Int. Immunopharmacol.

IF 4.8 2区 医学 Q2 IMMUNOLOGY
International immunopharmacology Pub Date : 2024-12-25 Epub Date: 2024-11-20 DOI:10.1016/j.intimp.2024.113557
Ehab A M El-Shoura, Ahmed A N Mohamed, Ahmed M Atwa, Esraa A Salem, Souty M Z Sharkawi, Hend Mostafa Selim, Aya Ibrahim Elberri, El-Sayed Gawesh, Yasmine H Ahmed, Omnia A M Abd El-Ghafar
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引用次数: 0

摘要

本文章由计算机程序翻译,如有差异,请以英文原文为准。
Corrigendum to "Combined diosmin and bisoprolol attenuate cobalt chloride-induced cardiotoxicity and endothelial dysfunction through modulating miR-143-3P/MAPK/MCP-1, ERK5/CXCR4, Orai-1/STIM-1 signaling pathways" [Int. Immunopharmacol. 140 (2024) 112777].
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来源期刊
CiteScore
8.40
自引率
3.60%
发文量
935
审稿时长
53 days
期刊介绍: International Immunopharmacology is the primary vehicle for the publication of original research papers pertinent to the overlapping areas of immunology, pharmacology, cytokine biology, immunotherapy, immunopathology and immunotoxicology. Review articles that encompass these subjects are also welcome. The subject material appropriate for submission includes: • Clinical studies employing immunotherapy of any type including the use of: bacterial and chemical agents; thymic hormones, interferon, lymphokines, etc., in transplantation and diseases such as cancer, immunodeficiency, chronic infection and allergic, inflammatory or autoimmune disorders. • Studies on the mechanisms of action of these agents for specific parameters of immune competence as well as the overall clinical state. • Pre-clinical animal studies and in vitro studies on mechanisms of action with immunopotentiators, immunomodulators, immunoadjuvants and other pharmacological agents active on cells participating in immune or allergic responses. • Pharmacological compounds, microbial products and toxicological agents that affect the lymphoid system, and their mechanisms of action. • Agents that activate genes or modify transcription and translation within the immune response. • Substances activated, generated, or released through immunologic or related pathways that are pharmacologically active. • Production, function and regulation of cytokines and their receptors. • Classical pharmacological studies on the effects of chemokines and bioactive factors released during immunological reactions.
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