回归:通过 SH3GL2 促进表皮生长因子受体(EGFR)下调,减少耐化疗 TNBC 的核 Y654-p-β-Catenin 表达:临床和预后的重要性。

IF 4.5 2区 生物学 Q2 CELL BIOLOGY
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引用次数: 0

摘要

撤回:S. Islam, H. Dasgupta, M. Basu, A. Roy, N. Alam, S. Roychoudhury, C. K. Panda, "Reduction of Nuclear Y654-p-β-Catenin Expression Through SH3GL2-Meditated Downregulation of EGFR in Chemotolerance TNBC:Clinical and Prognostic Importance," Journal of Cellular Physiology 235, no. 11 (2020): 8114-8128. https://doi.org/10.1002/jcp.29466。上述文章于 2020 年 1 月 20 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经期刊主编罗伯特-希思(Robert Heath)和 Wiley Periodicals LLC 协议撤回。该期刊收到了第三方关于本文图 2 C 和图 2 G 之间图像重复和旋转问题的通知,以及关于图 2 F 和图 3 C 被同一作者在另一篇文章(Islam, et al. 2020 [https://doi.org/10.1007/s13402-020-00525-5])中重复使用的问题。出版商的进一步调查也证实了本文中图 4 F 和补充图 1 C 与 1 F 之间的图像重复。在回答出版商的询问时,作者表示图 2 C 和图 2 G 之间的重复是由于分析了连续的组织切片。作者承认这篇文章和发表在不同期刊上的文章使用了相同的样本,但报告说他们在另一篇文章中错误地标注了样本。最后,作者表示,图 4 F 中以及补充图 1 C 和 1 F 之间的重复是由于制图过程中的失误造成的。双方同意,该解释无法解释本文内部以及本文与另一期刊发表的文章之间图片的多次重复和旋转。双方还同意,所提供的原始数据不足以支持作者的解释。同意撤稿的原因是,图像复制和篡改的证据从根本上损害了文章中提出的结论。作者不同意撤稿。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Reduction of Nuclear Y654-p-β-Catenin Expression Through SH3GL2-Meditated Downregulation of EGFR in Chemotolerance TNBC: Clinical and Prognostic Importance.

Retraction: S. Islam, H. Dasgupta, M. Basu, A. Roy, N. Alam, S. Roychoudhury, C. K. Panda, "Reduction of Nuclear Y654-p-β-Catenin Expression Through SH3GL2-Meditated Downregulation of EGFR in Chemotolerance TNBC: Clinical and Prognostic Importance," Journal of Cellular Physiology 235, no. 11 (2020): 8114-8128. https://doi.org/10.1002/jcp.29466. The above article, published online on 20 January 2020, in Wiley Online Library (wileyonlinelibrary.com), and has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. The journal received notice of concerns from a third party regarding duplications and rotation of images between Figures 2 C and 2 G in this article as well as concerns that Figures 2 F and 3 C had been re-used in a different article by some of the same authors (Islam, et al. 2020 [https://doi.org/10.1007/s13402-020-00525-5]). Additional investigation by the publisher also confirmed image duplications within Figure 4 F and between Supplementary Figures 1 C and 1 F in this article. In response to an inquiry by the publisher, the authors stated that the duplication between figures 2 C and 2 G was due to the fact that serial sections of tissue were analyzed. The authors acknowledged that the same samples had been used between this article and the article published in a different journal, but reported that they had mislabeled the samples in the other article. Lastly, the authors stated that the duplications in Figure 4 F and between Supplementary Figures 1 C and 1 F were caused by a mistake during figure preparation. The parties agree that the explanation does not account for the multiple duplications and rotations of images both within this article and between this article and the article published in another journal. The parties also agree that the original data as provided does not adequately support the authors' explanation. The retraction has been agreed to because the evidence of image duplication and manipulation fundamentally compromises the conclusions presented in the article. The authors disagree with the retraction.

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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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