跨膜 E3 连接酶 RNF128 通过促进核糖蛋白 I 泛素化和降解来调节 N-糖基化。

IF 2.9 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
BMB Reports Pub Date : 2024-12-01
Eun-Bee Cho, Van Anh Vu, Sang-Hee Park, Lan Thi Trinh, Jong-Bok Yoon, Sungjoo Kim
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引用次数: 0

摘要

环指蛋白128(RNF128)是一种跨膜E3泛素连接酶,主要定位于内质网,参与T细胞过敏和肿瘤进展等多种过程。然而,RNF128 在 N-糖基化过程中的生物学功能仍未被探索。为了探究RNF128的功能作用,我们使用了近位定向生物素标记法,发现核糖蛋白I(RPN1)是一种新型的RNF128底物,证明RNF128泛素化RPN1并促进其降解。RPN1 是寡糖基转移酶复合物的一个亚基,该复合物通过结合底物促进 N-糖基化,并将底物呈现给催化核心。RPN1 还是一种依赖于 N-糖基化的伴侣蛋白,有助于将新合成的糖蛋白子集导出到质膜。我们发现,RNF128 会影响模型糖蛋白的 N-糖基化,如性激素结合球蛋白和 Asialoglycoprotein receptor 1。此外,RNF128还抑制阿片受体μ1(OPRM1)向质膜的输出,而表达泛素化无能的RPN1突变体则能挽救RNF128过表达导致的OPRM1输出缺陷。此外,RNF128 还影响结直肠癌细胞的迁移。RNF128 依赖性降解 RPN1 可能会抑制特定糖蛋白在细胞表面的表达,从而影响不同的细胞功能。这项研究有助于理解 RNF128 和 RPN1 依赖性 N-糖基化的生物学和功能作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Transmembrane E3 ligase RNF128 regulates N-glycosylation by promoting ribophorin I ubiquitination and degradation.

Ring finger protein 128 (RNF128) is a transmembrane E3 ubiquitin ligase mainly localized in the endoplasmic reticulum that is involved in various processes, including T cell anergy and tumor progression. However, the biological function of RNF128 in N-glycosylation remains unexplored. To investigate the functional role of RNF128, we used the proximity-directed biotin labeling method, and identified ribophorin I (RPN1) as a novel RNF128 substrate, demonstrating that RNF128 ubiquitinated RPN1 and promoted its degradation. RPN1 is a subunit of oligosaccharyltransferase complexes that facilitate N-glycosylation by binding substrates, and presenting them to the catalytic core. RPN1 also functions as an N-glycosylation-dependent chaperone that helps export a subset of newly synthesized glycoproteins to the plasma membrane. We found that RNF128 affects the N-glycosylation of model glycoproteins, such as sex hormone- binding globulin and asialoglycoprotein receptor 1. Furthermore, RNF128 inhibits the export of the opioid receptor mu 1 (OPRM1) to the plasma membrane, while expressing ubiquitination-incompetent RPN1 mutant, rescues the defect of OPRM1 export caused by RNF128 overexpression. Additionally, RNF128 influences colorectal cancer cell migration. The RNF128-dependent degradation of RPN1 likely inhibits the cell surface expression of specific glycoproteins, thereby affecting distinct cellular functions. This study contributes to understanding of the biological and functional roles of RNF128- and RPN1-dependent N-glycosylation. [BMB Reports 2024; 57(12): 546-552].

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来源期刊
BMB Reports
BMB Reports 生物-生化与分子生物学
CiteScore
5.10
自引率
7.90%
发文量
141
审稿时长
1 months
期刊介绍: The BMB Reports (BMB Rep, established in 1968) is published at the end of every month by Korean Society for Biochemistry and Molecular Biology. Copyright is reserved by the Society. The journal publishes short articles and mini reviews. We expect that the BMB Reports will deliver the new scientific findings and knowledge to our readers in fast and timely manner.
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