磷酸化泛素对 E3 泛素连接酶 Parkin 催化核心的异构调控的合作亚结构和能量学研究

IF 4.8 2区 生物学 Q1 BIOCHEMISTRY & MOLECULAR BIOLOGY
Biomolecules Pub Date : 2024-10-21 DOI:10.3390/biom14101338
Xiang Ye, Sravya Kotaru, Rosana Lopes, Shannen Cravens, Mauricio Lasagna, A Joshua Wand
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引用次数: 0

摘要

Parkin 基因产品 Parkin 发生突变会导致常染色体隐性遗传的青少年帕金森病。Parkin 是一种 E3 泛素连接酶,是有丝分裂过程的重要参与者。Parkin 的结构复杂,整合了多种异构信号,以保持对其催化活性的精确控制。虽然晶体学已广泛描述了其异构控制的结构重组,但对 Parkin 异构调控的能量学和机制的了解却少得多。异构从根本上说与蛋白质的合作(亚)结构的能量学有关。在这里,我们研究了磷酸化泛素与缺少拮抗 Ubl 结构域的 Parkin 酶核心结合的异构激活机制。通过这种方法,可以分离出激动剂磷酸化泛素的异生效应。利用质谱监测的原生态氢交换,我们发现 Parkin 核心的五个结构域在能量上是不同的。然而,磷酸化泛素的结合会破坏结合泛素样结构域拮抗剂的结构元素的稳定性,同时促进催化结构域的解离,并在能量上使蛋白质过渡到完全活化的结构。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Cooperative Substructure and Energetics of Allosteric Regulation of the Catalytic Core of the E3 Ubiquitin Ligase Parkin by Phosphorylated Ubiquitin.

Mutations in the parkin gene product Parkin give rise to autosomal recessive juvenile parkinsonism. Parkin is an E3 ubiquitin ligase that is a critical participant in the process of mitophagy. Parkin has a complex structure that integrates several allosteric signals to maintain precise control of its catalytic activity. Though its allosterically controlled structural reorganization has been extensively characterized by crystallography, the energetics and mechanisms of allosteric regulation of Parkin are much less well understood. Allostery is fundamentally linked to the energetics of the cooperative (sub)structure of the protein. Herein, we examine the mechanism of allosteric activation by phosphorylated ubiquitin binding to the enzymatic core of Parkin, which lacks the antagonistic Ubl domain. In this way, the allosteric effects of the agonist phosphorylated ubiquitin can be isolated. Using native-state hydrogen exchange monitored by mass spectrometry, we find that the five structural domains of the core of Parkin are energetically distinct. Nevertheless, association of phosphorylated ubiquitin destabilizes structural elements that bind the ubiquitin-like domain antagonist while promoting the dissociation of the catalytic domain and energetically poises the protein for transition to the fully activated structure.

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来源期刊
Biomolecules
Biomolecules Biochemistry, Genetics and Molecular Biology-Molecular Biology
CiteScore
9.40
自引率
3.60%
发文量
1640
审稿时长
18.28 days
期刊介绍: Biomolecules (ISSN 2218-273X) is an international, peer-reviewed open access journal focusing on biogenic substances and their biological functions, structures, interactions with other molecules, and their microenvironment as well as biological systems. Biomolecules publishes reviews, regular research papers and short communications.  Our aim is to encourage scientists to publish their experimental and theoretical results in as much detail as possible. There is no restriction on the length of the papers. The full experimental details must be provided so that the results can be reproduced.
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