内皮细胞是长 COVID 的旁分泌介质

IF 9.4 Q1 CARDIAC & CARDIOVASCULAR SYSTEMS
Simon R. Foster, James E. Hudson
{"title":"内皮细胞是长 COVID 的旁分泌介质","authors":"Simon R. Foster, James E. Hudson","doi":"10.1038/s44161-024-00551-8","DOIUrl":null,"url":null,"abstract":"The molecular mechanisms that underpin the multi-organ dysfunction in long COVID are unknown, particularly within the cardiovascular system. Research finds a critical role for endothelial responses and signaling in driving dysfunction.","PeriodicalId":74245,"journal":{"name":"Nature cardiovascular research","volume":"3 10","pages":"1181-1183"},"PeriodicalIF":9.4000,"publicationDate":"2024-10-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Endothelial cells as paracrine mediators of long COVID\",\"authors\":\"Simon R. Foster, James E. Hudson\",\"doi\":\"10.1038/s44161-024-00551-8\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"The molecular mechanisms that underpin the multi-organ dysfunction in long COVID are unknown, particularly within the cardiovascular system. Research finds a critical role for endothelial responses and signaling in driving dysfunction.\",\"PeriodicalId\":74245,\"journal\":{\"name\":\"Nature cardiovascular research\",\"volume\":\"3 10\",\"pages\":\"1181-1183\"},\"PeriodicalIF\":9.4000,\"publicationDate\":\"2024-10-14\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Nature cardiovascular research\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"https://www.nature.com/articles/s44161-024-00551-8\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q1\",\"JCRName\":\"CARDIAC & CARDIOVASCULAR SYSTEMS\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Nature cardiovascular research","FirstCategoryId":"1085","ListUrlMain":"https://www.nature.com/articles/s44161-024-00551-8","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q1","JCRName":"CARDIAC & CARDIOVASCULAR SYSTEMS","Score":null,"Total":0}
引用次数: 0

摘要

长期 COVID 导致多器官功能障碍的分子机制尚不清楚,尤其是心血管系统。研究发现,内皮反应和信号传导在驱动功能障碍方面起着关键作用。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Endothelial cells as paracrine mediators of long COVID

Endothelial cells as paracrine mediators of long COVID
The molecular mechanisms that underpin the multi-organ dysfunction in long COVID are unknown, particularly within the cardiovascular system. Research finds a critical role for endothelial responses and signaling in driving dysfunction.
求助全文
通过发布文献求助,成功后即可免费获取论文全文。 去求助
来源期刊
CiteScore
5.70
自引率
0.00%
发文量
0
×
引用
GB/T 7714-2015
复制
MLA
复制
APA
复制
导出至
BibTeX EndNote RefMan NoteFirst NoteExpress
×
提示
您的信息不完整,为了账户安全,请先补充。
现在去补充
×
提示
您因"违规操作"
具体请查看互助需知
我知道了
×
提示
确定
请完成安全验证×
copy
已复制链接
快去分享给好友吧!
我知道了
右上角分享
点击右上角分享
0
联系我们:info@booksci.cn Book学术提供免费学术资源搜索服务,方便国内外学者检索中英文文献。致力于提供最便捷和优质的服务体验。 Copyright © 2023 布克学术 All rights reserved.
京ICP备2023020795号-1
ghs 京公网安备 11010802042870号
Book学术文献互助
Book学术文献互助群
群 号:481959085
Book学术官方微信