Kevin Halter, Jingyi Chen, Tadeas Priklopil, Asun Monfort, Anton Wutz
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引用次数: 0
摘要
小鼠胚胎干细胞具有多能发育潜能和稳定的核型。一个例外是,近交系小鼠的雌性胚胎干细胞经常丢失一条X染色体。相反,来自不同麝亚种杂交的雌性 ESCs 通常保持两条 X 染色体,并能模拟 X 染色体失活。在这里,我们报告了在一个原本保持两条X染色体的蓖麻麝杂交雌性ESC品系中,Hira和Cdk8的联合突变诱导了一条X染色体的快速缺失。我们发现,用于培养幼稚多能 ESCs 的 MEK1 抑制足以诱导 X 染色体缺失。在传统的造血干细胞培养基中,Hira和Cdk8突变型造血干细胞能保持两条X染色体。通过改用天真培养基诱导X染色体缺失,我们可以进行动力学测量,计算染色体缺失率。我们的分析表明,X染色体缺失的原因不是XO细胞的选择,而可能是染色体的消除过程。我们发现,X 染色体的消除率为每个细胞每次分裂的 0.3%,比报告的常染色体丢失率高出 3 个数量级。我们发现,8 号和 11 号染色体得到了稳定的维持。值得注意的是,来自两条X染色体之一的Xist表达能挽救ΔHiraΔCdk8 ESC中X染色体的不稳定性。我们的研究确定了Hira和Cdk8的突变是导致天真雌性ESC中X染色体消除的分子驱动因素,并描述了一种用于阐明潜在机制的细胞系统。
Cdk8 and Hira mutations trigger X chromosome elimination in naive female hybrid mouse embryonic stem cells.
Mouse embryonic stem cells (ESCs) possess a pluripotent developmental potential and a stable karyotype. An exception is the frequent loss of one X chromosome in female ESCs derived from inbred mice. In contrast, female ESCs from crosses between different Mus musculus subspecies often maintain two X chromosomes and can model X chromosome inactivation. Here we report that combined mutations of Hira and Cdk8 induce rapid loss of one X chromosome in a Mus musculus castaneus hybrid female ESC line that originally maintains two X chromosomes. We show that MEK1 inhibition, which is used for culturing naive pluripotent ESCs is sufficient to induce X chromosome loss. In conventional ESC media, Hira and Cdk8 mutant ESCs maintain both X chromosomes. Induction of X chromosome loss by switching to naive culture media allows us to perform kinetic measurements for calculating the chromosome loss rate. Our analysis shows that X chromosome loss is not explained by selection of XO cells, but likely driven by a process of chromosome elimination. We show that elimination of the X chromosome occurs with a rate of 0.3% per cell per division, which exceeds reported autosomal loss rates by 3 orders of magnitude. We show that chromosomes 8 and 11 are stably maintained. Notably, Xist expression from one of the two X chromosomes rescues X chromosomal instability in ΔHiraΔCdk8 ESCs. Our study defines mutations of Hira and Cdk8 as molecular drivers for X chromosome elimination in naive female ESCs and describes a cell system for elucidating the underlying mechanism.
期刊介绍:
Chromosome Research publishes manuscripts from work based on all organisms and encourages submissions in the following areas including, but not limited, to:
· Chromosomes and their linkage to diseases;
· Chromosome organization within the nucleus;
· Chromatin biology (transcription, non-coding RNA, etc);
· Chromosome structure, function and mechanics;
· Chromosome and DNA repair;
· Epigenetic chromosomal functions (centromeres, telomeres, replication, imprinting,
dosage compensation, sex determination, chromosome remodeling);
· Architectural/epigenomic organization of the genome;
· Functional annotation of the genome;
· Functional and comparative genomics in plants and animals;
· Karyology studies that help resolve difficult taxonomic problems or that provide
clues to fundamental mechanisms of genome and karyotype evolution in plants and animals;
· Mitosis and Meiosis;
· Cancer cytogenomics.