回放:环境对昼夜节律的干扰易使小鼠膝关节发生骨关节炎样改变。

IF 4.5 2区 生物学 Q2 CELL BIOLOGY
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引用次数: 0

摘要

撤回:R.Kc、X.Li、R.M.Voigt、M.B.Ellman、K.C.Summa、M.H.Vitaterna、A.Keshavarizian、F.W.Turek、Q.-J.Meng、G.S.Stein、A.J.van Wijnen、D.Chen、C.B.Forsyth 和 H.J.Im, "Environmental Disruption Circadian Rhythm Predisposes Mice to Osteoarthritis-Like Changes in Knee Joint," Journal of Cellular Physiology 230.J. Im, "Environmental Disruption of Circadian Rhythm Predisposes Mice to Osteoarthritis-Like Changes in Knee Joint," Journal of Cellular Physiology 230, no. 9 (2015): 2174-2183, https://doi.org/10.1002/jcp.24946.上述文章于 2015 年 2 月 5 日在线发表于 Wiley Online Library (wileyonlinelibrary.com),经期刊主编罗伯特-希思(Robert Heath)和 Wiley Periodicals LLC 协议,该文章已被撤回。撤稿的原因是拉什大学医学中心研究合规办公室在拉什大学和杰西-布朗退伍军人事务医疗中心(JBVAMC)联合开展调查后,对文章中提供的数据提出了质疑。具体而言,图 4 A 中实验数据的图像元素被发现已被同一作者在不同的科学背景下用于在其他地方发表。通讯作者 Hee-Jeong Im Sampen 博士已被告知撤稿决定,但她并不同意撤稿,因为她相信论文中的任何错误都不会影响论文研究结果的可靠性。她还告知编辑,她随时准备全力配合进行必要的更正。然而,由于编辑们对数据的准确性失去了信任,并认为结论无效,因此文章被撤回。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
RETRACTION: Environmental Disruption of Circadian Rhythm Predisposes Mice to Osteoarthritis-Like Changes in Knee Joint.

Retraction: R. Kc, X. Li, R. M. Voigt, M. B. Ellman, K. C. Summa, M. H. Vitaterna, A. Keshavarizian, F. W. Turek, Q.-J. Meng, G. S. Stein, A. J. van Wijnen, D. Chen, C. B. Forsyth, and H.-J. Im, "Environmental Disruption of Circadian Rhythm Predisposes Mice to Osteoarthritis-Like Changes in Knee Joint," Journal of Cellular Physiology 230, no. 9 (2015): 2174-2183, https://doi.org/10.1002/jcp.24946. The above article, published online on 5 February 2015 in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement between the journal Editor-in-Chief, Robert Heath; and Wiley Periodicals LLC. The retraction has occurred due to concerns related to the data presented in the article raised by the Office of Research Compliance at Rush University Medical Center following an investigation jointly conducted by Rush University and the Jesse Brown Veterans Affairs Medical Center (JBVAMC). Specifically, image elements of the experimental data in Figure 4 A were found to have been used by the same author(s) for publication elsewhere in a different scientific context. The corresponding author, Dr. Hee-Jeong Im Sampen, has been informed of the decision to retract but did not agree with it, as she is confident that any errors in the publication do not impact the reliability of the paper's findings. She also advised the editors that she stands ready to cooperate fully to make any necessary corrections. However, the article is retracted as the editors lost trust in the accuracy of the data and consider the conclusions invalid.

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来源期刊
CiteScore
14.70
自引率
0.00%
发文量
256
审稿时长
1 months
期刊介绍: The Journal of Cellular Physiology publishes reports of high biological significance in areas of eukaryotic cell biology and physiology, focusing on those articles that adopt a molecular mechanistic approach to investigate cell structure and function. There is appreciation for the application of cellular, biochemical, molecular and in vivo genetic approaches, as well as the power of genomics, proteomics, bioinformatics and systems biology. In particular, the Journal encourages submission of high-interest papers investigating the genetic and epigenetic regulation of proliferation and phenotype as well as cell fate and lineage commitment by growth factors, cytokines and their cognate receptors and signal transduction pathways that influence the expression, integration and activities of these physiological mediators. Similarly, the Journal encourages submission of manuscripts exploring the regulation of growth and differentiation by cell adhesion molecules in addition to the interplay between these processes and those induced by growth factors and cytokines. Studies on the genes and processes that regulate cell cycle progression and phase transition in eukaryotic cells, and the mechanisms that determine whether cells enter quiescence, proliferate or undergo apoptosis are also welcomed. Submission of papers that address contributions of the extracellular matrix to cellular phenotypes and physiological control as well as regulatory mechanisms governing fertilization, embryogenesis, gametogenesis, cell fate, lineage commitment, differentiation, development and dynamic parameters of cell motility are encouraged. Finally, the investigation of stem cells and changes that differentiate cancer cells from normal cells including studies on the properties and functions of oncogenes and tumor suppressor genes will remain as one of the major interests of the Journal.
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