[撤稿】MicroRNA-519 通过降低 RNA 结合蛋白人类抗原 R 的表达水平增强 HL60 人类急性髓性白血病细胞系的增殖。

IF 3.4 3区 医学 Q2 MEDICINE, RESEARCH & EXPERIMENTAL
Molecular medicine reports Pub Date : 2024-12-01 Epub Date: 2024-09-27 DOI:10.3892/mmr.2024.13335
Kebin Huang, Bingwei Dong, Yueyue Wang, Tao Tian, Biying Zhang
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引用次数: 0

摘要

这篇论文发表后,一位相关读者提请编辑注意,第 7834 页图 4B 中显示的某些流式细胞分析数据与已以不同形式提交发表的另一篇文章中的数据惊人相似,而这篇文章是由不同研究机构的不同作者撰写的。由于上述文章中有争议的数据在提交给《分子医学报告》之前就已经提交发表,因此编辑决定从《分子医学报告》上撤回这篇论文。编辑部要求作者就这些问题做出解释,但未收到回复。对于给读者带来的不便,编辑深表歉意。[分子医学报告 12: 7830-7836, 2015; DOI: 10.3892/mmr.2015.4455]。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Retracted] MicroRNA‑519 enhances HL60 human acute myeloid leukemia cell line proliferation by reducing the expression level of RNA‑binding protein human antigen R.

Following the publication of this paper, it was drawn to the Editor's attention by a concerned reader that certain of the flow cytometric analysis data shown in Fig. 4B on p. 7834 were strikingly similar to data that had already been submitted for publication in different form in another article written by different authors at different research institutes. Owing to the fact that the contentious data in the above article had already been submitted for publication prior to its submission to Molecular Medicine Reports, the Editor has decided that this paper should be retracted from the Journal. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a reply. The Editor apologizes to the readership for any inconvenience caused. [Molecular Medicine Reports 12: 7830‑7836, 2015; DOI: 10.3892/mmr.2015.4455].

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来源期刊
Molecular medicine reports
Molecular medicine reports 医学-病理学
CiteScore
7.60
自引率
0.00%
发文量
321
审稿时长
1.5 months
期刊介绍: Molecular Medicine Reports is a monthly, peer-reviewed journal available in print and online, that includes studies devoted to molecular medicine, underscoring aspects including pharmacology, pathology, genetics, neurosciences, infectious diseases, molecular cardiology and molecular surgery. In vitro and in vivo studies of experimental model systems pertaining to the mechanisms of a variety of diseases offer researchers the necessary tools and knowledge with which to aid the diagnosis and treatment of human diseases.
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