前脑 Eml1 缺失揭示了导致皮层下异位的早期中心粒功能障碍。

IF 7.4 1区 生物学 Q1 CELL BIOLOGY
Donia Zaidi,Kaviya Chinnappa,Berfu Nur Yigit,Valeria Viola,Carmen Cifuentes-Diaz,Ammar Jabali,Ana Uzquiano,Emilie Lemesre,Franck Perez,Julia Ladewig,Julien Ferent,Nurhan Ozlu,Fiona Francis
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引用次数: 0

摘要

皮层下异位症是一种与癫痫、智力障碍和白质中皮层神经元数量过多有关的皮层畸形。棘皮动物微管相关蛋白 1(EML1)突变导致皮层下异位症,在畸形发生之前,皮层壁上的放射状胶质细胞定位异常。这种增殖细胞分布的紊乱可能是异位畸形形成的关键事件;然而,其潜在机制仍未解释。本研究旨在破译导致异常放射状胶质细胞的早期细胞改变。在前脑条件性 Eml1 突变模型和人类患者细胞中,初级纤毛和中心体发生了改变。小鼠突变型放射状胶质细胞的微管动力学和细胞周期动力学也出现异常。通过挽救 Eml1 突变体胚胎大脑中的微管形成,异常的放射状胶质细胞分层和异位细胞体积显著减少。因此,我们的皮层下异位症新模型揭示了 Eml1 在微管调控和细胞位置功能之间的因果联系,而这两种功能对于皮层的正确发育都至关重要。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Forebrain Eml1 depletion reveals early centrosomal dysfunction causing subcortical heterotopia.
Subcortical heterotopia is a cortical malformation associated with epilepsy, intellectual disability, and an excessive number of cortical neurons in the white matter. Echinoderm microtubule-associated protein like 1 (EML1) mutations lead to subcortical heterotopia, associated with abnormal radial glia positioning in the cortical wall, prior to malformation onset. This perturbed distribution of proliferative cells is likely to be a critical event for heterotopia formation; however, the underlying mechanisms remain unexplained. This study aimed to decipher the early cellular alterations leading to abnormal radial glia. In a forebrain conditional Eml1 mutant model and human patient cells, primary cilia and centrosomes are altered. Microtubule dynamics and cell cycle kinetics are also abnormal in mouse mutant radial glia. By rescuing microtubule formation in Eml1 mutant embryonic brains, abnormal radial glia delamination and heterotopia volume were significantly reduced. Thus, our new model of subcortical heterotopia reveals the causal link between Eml1's function in microtubule regulation and cell position, both critical for correct cortical development.
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来源期刊
Journal of Cell Biology
Journal of Cell Biology 生物-细胞生物学
CiteScore
12.60
自引率
2.60%
发文量
213
审稿时长
1 months
期刊介绍: The Journal of Cell Biology (JCB) is a comprehensive journal dedicated to publishing original discoveries across all realms of cell biology. We invite papers presenting novel cellular or molecular advancements in various domains of basic cell biology, along with applied cell biology research in diverse systems such as immunology, neurobiology, metabolism, virology, developmental biology, and plant biology. We enthusiastically welcome submissions showcasing significant findings of interest to cell biologists, irrespective of the experimental approach.
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