Stephan Sachs, Anna Götz, Brian Finan, Annette Feuchtinger, Richard D. DiMarchi, Yvonne Döring, Christian Weber, Matthias H. Tschöp, Timo D. Müller, Susanna M. Hofmann
{"title":"更正:在心血管代谢疾病临床前小鼠模型中,GIP 受体激动可改善血脂异常和动脉粥样硬化,而与体重减轻无关","authors":"Stephan Sachs, Anna Götz, Brian Finan, Annette Feuchtinger, Richard D. DiMarchi, Yvonne Döring, Christian Weber, Matthias H. Tschöp, Timo D. Müller, Susanna M. Hofmann","doi":"10.1186/s12933-024-02407-8","DOIUrl":null,"url":null,"abstract":"<p><b>Correction to: Cardiovasc Diabetol (2023) 22:217</b></p><p>https://doi.org/10.1186/s12933-023-01940-2</p><p>Following publication of the original article [1], the author noticed the errors in Fig. 2 and in Results section.</p><p>The bar graph is mistakenly duplicated in “percentage of plaque area of the aortic valves” of Fig. 2E. The corrected figure is given below:</p><figure><figcaption><b data-test=\"figure-caption-text\">Fig. 2</b></figcaption><picture><source srcset=\"//media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12933-024-02407-8/MediaObjects/12933_2024_2407_Fig2_HTML.png?as=webp\" type=\"image/webp\"/><img alt=\"figure 2\" aria-describedby=\"Fig2\" height=\"344\" loading=\"lazy\" src=\"//media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12933-024-02407-8/MediaObjects/12933_2024_2407_Fig2_HTML.png\" width=\"685\"/></picture><p>Acyl-GIP ameliorates dyslipidemia and atherosclerotic plaque formation in LDLR-/- male mice. Plasma (<b>A</b>) triglycerides, (<b>B</b>) cholesterol and (<b>C</b>) lipoprotein fractions as well as (<b>D</b> and <b>E</b>) the percentage of plaque area in aortic arches and valves and along the descending aorta of male LDLR-/- mice treated daily with either vehicle or acyl-GIP via subcutaneous injections for 28 days. <i>n</i> = 7. Blood lipids were determined from sac plasma at the end of the study. Data represent means ± SEM. *<i>P</i> < 0.05, **<i>P</i> < 0.01, *** <i>P</i> < 0.001, determined by unpaired two-sided t-test</p><span>Full size image</span><svg aria-hidden=\"true\" focusable=\"false\" height=\"16\" role=\"img\" width=\"16\"><use xlink:href=\"#icon-eds-i-chevron-right-small\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"></use></svg></figure><p>In Result section under the heading “GIPR-agonist acyl-GIP ameliorates dyslipidemia and atherosclerotic plaque formation in male LDLR-/- mice independently of weight loss”, the last sentence should read “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve and a trend to decrease fat streaks along the descending aorta (Fig. 2E)“ instead of “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve (Fig. 2G–H) and decreased fat streaks along the descending aorta (Fig. 2I)”.</p><ol data-track-component=\"outbound reference\" data-track-context=\"references section\"><li data-counter=\"1.\"><p>Sachs S, Götz A, Finan B, et al. GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. Cardiovasc Diabetol. 2023;22:217.https://doi.org/10.1186/s12933-023-01940-2</p><p>Article CAS PubMed PubMed Central Google Scholar </p></li></ol><p>Download references<svg aria-hidden=\"true\" focusable=\"false\" height=\"16\" role=\"img\" width=\"16\"><use xlink:href=\"#icon-eds-i-download-medium\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"></use></svg></p><span>Author notes</span><ol><li><p>Stephan Sachs and Anna Götz contributed equally.</p></li></ol><h3>Authors and Affiliations</h3><ol><li><p>Institute for Diabetes and Regeneration, Helmholtz Diabetes Center at Helmholtz Zentrum München, German Research Center for Environmental Health (GmbH), 85764, Neuherberg, Germany</p><p>Stephan Sachs, Anna Götz & Susanna M. Hofmann</p></li><li><p>Institute for Diabetes and Obesity, Division of Metabolic Diseases, Helmholtz Diabetes Center at Helmholtz Centre Munich, Munich, Germany</p><p>Stephan Sachs, Anna Götz, Matthias H. Tschöp & Timo D. Müller</p></li><li><p>Technische Universität München, 80333, Munich, Germany</p><p>Stephan Sachs & Matthias H. Tschöp</p></li><li><p>Novo Nordisk Research Center Indianapolis, Indianapolis, IN, USA</p><p>Brian Finan</p></li><li><p>Research Unit Analytical Pathology, Helmholtz Center Munich, 85764, Neuherberg, Germany</p><p>Annette Feuchtinger</p></li><li><p>Department of Chemistry, Indiana University, Bloomington, IN, USA</p><p>Richard D. DiMarchi</p></li><li><p>Department of Angiology, Swiss Cardiovascular Center, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland</p><p>Yvonne Döring</p></li><li><p>Institute for Cardiovascular Prevention (IPEK), Ludwig-Maximilians-University Munich, Munich, Germany</p><p>Yvonne Döring & Christian Weber</p></li><li><p>DZHK (German Centre for Cardiovascular Research), Partner Site Munich Heart Alliance, Munich, Germany</p><p>Yvonne Döring & Christian Weber</p></li><li><p>Department of Biochemistry, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, the Netherlands</p><p>Christian Weber</p></li><li><p>Munich Cluster for Systems Neurology (SyNergy), Munich, Germany</p><p>Christian Weber</p></li><li><p>German Center for Diabetes Research (DZD), 85764, Neuherberg, Germany</p><p>Matthias H. Tschöp, Timo D. Müller & Susanna M. Hofmann</p></li><li><p>Department of Medicine IV, University Hospital, LMU Munich, Munich, Germany</p><p>Susanna M. Hofmann</p></li></ol><span>Authors</span><ol><li><span>Stephan Sachs</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Anna Götz</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Brian Finan</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Annette Feuchtinger</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Richard D. DiMarchi</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Yvonne Döring</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Christian Weber</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Matthias H. Tschöp</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Timo D. Müller</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Susanna M. Hofmann</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li></ol><h3>Corresponding authors</h3><p>Correspondence to Timo D. Müller or Susanna M. Hofmann.</p><h3>Publisher’s note</h3><p>Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.</p><p><b>Open Access</b> This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. 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0-.92-.08-1.33-.25-.41-.16-.77-.4-1.08-.7-.3-.31-.54-.69-.72-1.13-.17-.44-.26-.95-.26-1.52zm4.61-.62c0-.55-.11-.98-.34-1.28-.23-.31-.58-.47-1.06-.47-.41 0-.77.15-1.08.45-.31.29-.5.73-.57 1.3zm3.01 2.23c.31.24.61.43.92.57.3.13.63.2.98.2.38 0 .65-.08.83-.23s.27-.35.27-.6c0-.14-.05-.26-.13-.37-.08-.1-.2-.2-.34-.28-.14-.09-.29-.16-.47-.23l-.53-.22c-.23-.09-.46-.18-.69-.3-.23-.11-.44-.24-.62-.4s-.33-.35-.45-.55c-.12-.21-.18-.46-.18-.75 0-.61.23-1.1.68-1.49.44-.38 1.06-.57 1.83-.57.48 0 .91.08 1.29.25s.71.36.99.57l-.74.98c-.24-.17-.49-.32-.73-.42-.25-.11-.51-.16-.78-.16-.35 0-.6.07-.76.21-.17.15-.25.33-.25.54 0 .14.04.26.12.36s.18.18.31.26c.14.07.29.14.46.21l.54.19c.23.09.47.18.7.29s.44.24.64.4c.19.16.34.35.46.58.11.23.17.5.17.82 0 .3-.06.58-.17.83-.12.26-.29.48-.51.68-.23.19-.51.34-.84.45-.34.11-.72.17-1.15.17-.48 0-.95-.09-1.41-.27-.46-.19-.86-.41-1.2-.68z" fill="#535353"/></g></svg>\" width=\"57\"/><h3>Cite this article</h3><p>Sachs, S., Götz, A., Finan, B. <i>et al.</i> Correction: GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. <i>Cardiovasc Diabetol</i> <b>23</b>, 341 (2024). https://doi.org/10.1186/s12933-024-02407-8</p><p>Download citation<svg aria-hidden=\"true\" focusable=\"false\" height=\"16\" role=\"img\" width=\"16\"><use xlink:href=\"#icon-eds-i-download-medium\" xmlns:xlink=\"http://www.w3.org/1999/xlink\"></use></svg></p><ul data-test=\"publication-history\"><li><p>Published<span>: </span><span><time datetime=\"2024-09-12\">12 September 2024</time></span></p></li><li><p>DOI</abbr><span>: </span><span>https://doi.org/10.1186/s12933-024-02407-8</span></p></li></ul><h3>Share this article</h3><p>Anyone you share the following link with will be able to read this content:</p><button data-track=\"click\" data-track-action=\"get shareable link\" data-track-external=\"\" data-track-label=\"button\" type=\"button\">Get shareable link</button><p>Sorry, a shareable link is not currently available for this article.</p><p data-track=\"click\" data-track-action=\"select share url\" data-track-label=\"button\"></p><button data-track=\"click\" data-track-action=\"copy share url\" data-track-external=\"\" data-track-label=\"button\" type=\"button\">Copy to clipboard</button><p> Provided by the Springer Nature SharedIt content-sharing initiative </p>","PeriodicalId":9374,"journal":{"name":"Cardiovascular Diabetology","volume":"18 1","pages":""},"PeriodicalIF":8.5000,"publicationDate":"2024-09-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"Correction: GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease\",\"authors\":\"Stephan Sachs, Anna Götz, Brian Finan, Annette Feuchtinger, Richard D. DiMarchi, Yvonne Döring, Christian Weber, Matthias H. Tschöp, Timo D. Müller, Susanna M. Hofmann\",\"doi\":\"10.1186/s12933-024-02407-8\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><b>Correction to: Cardiovasc Diabetol (2023) 22:217</b></p><p>https://doi.org/10.1186/s12933-023-01940-2</p><p>Following publication of the original article [1], the author noticed the errors in Fig. 2 and in Results section.</p><p>The bar graph is mistakenly duplicated in “percentage of plaque area of the aortic valves” of Fig. 2E. The corrected figure is given below:</p><figure><figcaption><b data-test=\\\"figure-caption-text\\\">Fig. 2</b></figcaption><picture><source srcset=\\\"//media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12933-024-02407-8/MediaObjects/12933_2024_2407_Fig2_HTML.png?as=webp\\\" type=\\\"image/webp\\\"/><img alt=\\\"figure 2\\\" aria-describedby=\\\"Fig2\\\" height=\\\"344\\\" loading=\\\"lazy\\\" src=\\\"//media.springernature.com/lw685/springer-static/image/art%3A10.1186%2Fs12933-024-02407-8/MediaObjects/12933_2024_2407_Fig2_HTML.png\\\" width=\\\"685\\\"/></picture><p>Acyl-GIP ameliorates dyslipidemia and atherosclerotic plaque formation in LDLR-/- male mice. Plasma (<b>A</b>) triglycerides, (<b>B</b>) cholesterol and (<b>C</b>) lipoprotein fractions as well as (<b>D</b> and <b>E</b>) the percentage of plaque area in aortic arches and valves and along the descending aorta of male LDLR-/- mice treated daily with either vehicle or acyl-GIP via subcutaneous injections for 28 days. <i>n</i> = 7. Blood lipids were determined from sac plasma at the end of the study. Data represent means ± SEM. *<i>P</i> < 0.05, **<i>P</i> < 0.01, *** <i>P</i> < 0.001, determined by unpaired two-sided t-test</p><span>Full size image</span><svg aria-hidden=\\\"true\\\" focusable=\\\"false\\\" height=\\\"16\\\" role=\\\"img\\\" width=\\\"16\\\"><use xlink:href=\\\"#icon-eds-i-chevron-right-small\\\" xmlns:xlink=\\\"http://www.w3.org/1999/xlink\\\"></use></svg></figure><p>In Result section under the heading “GIPR-agonist acyl-GIP ameliorates dyslipidemia and atherosclerotic plaque formation in male LDLR-/- mice independently of weight loss”, the last sentence should read “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve and a trend to decrease fat streaks along the descending aorta (Fig. 2E)“ instead of “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve (Fig. 2G–H) and decreased fat streaks along the descending aorta (Fig. 2I)”.</p><ol data-track-component=\\\"outbound reference\\\" data-track-context=\\\"references section\\\"><li data-counter=\\\"1.\\\"><p>Sachs S, Götz A, Finan B, et al. GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. Cardiovasc Diabetol. 2023;22:217.https://doi.org/10.1186/s12933-023-01940-2</p><p>Article CAS PubMed PubMed Central Google Scholar </p></li></ol><p>Download references<svg aria-hidden=\\\"true\\\" focusable=\\\"false\\\" height=\\\"16\\\" role=\\\"img\\\" width=\\\"16\\\"><use xlink:href=\\\"#icon-eds-i-download-medium\\\" xmlns:xlink=\\\"http://www.w3.org/1999/xlink\\\"></use></svg></p><span>Author notes</span><ol><li><p>Stephan Sachs and Anna Götz contributed equally.</p></li></ol><h3>Authors and Affiliations</h3><ol><li><p>Institute for Diabetes and Regeneration, Helmholtz Diabetes Center at Helmholtz Zentrum München, German Research Center for Environmental Health (GmbH), 85764, Neuherberg, Germany</p><p>Stephan Sachs, Anna Götz & Susanna M. Hofmann</p></li><li><p>Institute for Diabetes and Obesity, Division of Metabolic Diseases, Helmholtz Diabetes Center at Helmholtz Centre Munich, Munich, Germany</p><p>Stephan Sachs, Anna Götz, Matthias H. Tschöp & Timo D. Müller</p></li><li><p>Technische Universität München, 80333, Munich, Germany</p><p>Stephan Sachs & Matthias H. Tschöp</p></li><li><p>Novo Nordisk Research Center Indianapolis, Indianapolis, IN, USA</p><p>Brian Finan</p></li><li><p>Research Unit Analytical Pathology, Helmholtz Center Munich, 85764, Neuherberg, Germany</p><p>Annette Feuchtinger</p></li><li><p>Department of Chemistry, Indiana University, Bloomington, IN, USA</p><p>Richard D. DiMarchi</p></li><li><p>Department of Angiology, Swiss Cardiovascular Center, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland</p><p>Yvonne Döring</p></li><li><p>Institute for Cardiovascular Prevention (IPEK), Ludwig-Maximilians-University Munich, Munich, Germany</p><p>Yvonne Döring & Christian Weber</p></li><li><p>DZHK (German Centre for Cardiovascular Research), Partner Site Munich Heart Alliance, Munich, Germany</p><p>Yvonne Döring & Christian Weber</p></li><li><p>Department of Biochemistry, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, the Netherlands</p><p>Christian Weber</p></li><li><p>Munich Cluster for Systems Neurology (SyNergy), Munich, Germany</p><p>Christian Weber</p></li><li><p>German Center for Diabetes Research (DZD), 85764, Neuherberg, Germany</p><p>Matthias H. Tschöp, Timo D. Müller & Susanna M. Hofmann</p></li><li><p>Department of Medicine IV, University Hospital, LMU Munich, Munich, Germany</p><p>Susanna M. Hofmann</p></li></ol><span>Authors</span><ol><li><span>Stephan Sachs</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Anna Götz</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Brian Finan</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Annette Feuchtinger</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Richard D. DiMarchi</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Yvonne Döring</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Christian Weber</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Matthias H. Tschöp</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Timo D. Müller</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li><li><span>Susanna M. Hofmann</span>View author publications<p>You can also search for this author in <span>PubMed<span> </span>Google Scholar</span></p></li></ol><h3>Corresponding authors</h3><p>Correspondence to Timo D. Müller or Susanna M. 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GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. <i>Cardiovasc Diabetol</i> <b>23</b>, 341 (2024). https://doi.org/10.1186/s12933-024-02407-8</p><p>Download citation<svg aria-hidden=\\\"true\\\" focusable=\\\"false\\\" height=\\\"16\\\" role=\\\"img\\\" width=\\\"16\\\"><use xlink:href=\\\"#icon-eds-i-download-medium\\\" xmlns:xlink=\\\"http://www.w3.org/1999/xlink\\\"></use></svg></p><ul data-test=\\\"publication-history\\\"><li><p>Published<span>: </span><span><time datetime=\\\"2024-09-12\\\">12 September 2024</time></span></p></li><li><p>DOI</abbr><span>: </span><span>https://doi.org/10.1186/s12933-024-02407-8</span></p></li></ul><h3>Share this article</h3><p>Anyone you share the following link with will be able to read this content:</p><button data-track=\\\"click\\\" data-track-action=\\\"get shareable link\\\" data-track-external=\\\"\\\" data-track-label=\\\"button\\\" 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Correction: GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease
Correction to: Cardiovasc Diabetol (2023) 22:217
https://doi.org/10.1186/s12933-023-01940-2
Following publication of the original article [1], the author noticed the errors in Fig. 2 and in Results section.
The bar graph is mistakenly duplicated in “percentage of plaque area of the aortic valves” of Fig. 2E. The corrected figure is given below:
In Result section under the heading “GIPR-agonist acyl-GIP ameliorates dyslipidemia and atherosclerotic plaque formation in male LDLR-/- mice independently of weight loss”, the last sentence should read “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve and a trend to decrease fat streaks along the descending aorta (Fig. 2E)“ instead of “Most importantly, acyl-GIP treatment was accompanied by reduced atherosclerotic plaque formation within the aortic valve (Fig. 2G–H) and decreased fat streaks along the descending aorta (Fig. 2I)”.
Sachs S, Götz A, Finan B, et al. GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. Cardiovasc Diabetol. 2023;22:217.https://doi.org/10.1186/s12933-023-01940-2
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Author notes
Stephan Sachs and Anna Götz contributed equally.
Authors and Affiliations
Institute for Diabetes and Regeneration, Helmholtz Diabetes Center at Helmholtz Zentrum München, German Research Center for Environmental Health (GmbH), 85764, Neuherberg, Germany
Stephan Sachs, Anna Götz & Susanna M. Hofmann
Institute for Diabetes and Obesity, Division of Metabolic Diseases, Helmholtz Diabetes Center at Helmholtz Centre Munich, Munich, Germany
Stephan Sachs, Anna Götz, Matthias H. Tschöp & Timo D. Müller
Technische Universität München, 80333, Munich, Germany
Stephan Sachs & Matthias H. Tschöp
Novo Nordisk Research Center Indianapolis, Indianapolis, IN, USA
Brian Finan
Research Unit Analytical Pathology, Helmholtz Center Munich, 85764, Neuherberg, Germany
Annette Feuchtinger
Department of Chemistry, Indiana University, Bloomington, IN, USA
Richard D. DiMarchi
Department of Angiology, Swiss Cardiovascular Center, Inselspital, Bern University Hospital, University of Bern, Bern, Switzerland
Yvonne Döring
Institute for Cardiovascular Prevention (IPEK), Ludwig-Maximilians-University Munich, Munich, Germany
Yvonne Döring & Christian Weber
DZHK (German Centre for Cardiovascular Research), Partner Site Munich Heart Alliance, Munich, Germany
Yvonne Döring & Christian Weber
Department of Biochemistry, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, Maastricht, the Netherlands
Christian Weber
Munich Cluster for Systems Neurology (SyNergy), Munich, Germany
Christian Weber
German Center for Diabetes Research (DZD), 85764, Neuherberg, Germany
Matthias H. Tschöp, Timo D. Müller & Susanna M. Hofmann
Department of Medicine IV, University Hospital, LMU Munich, Munich, Germany
Susanna M. Hofmann
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Sachs, S., Götz, A., Finan, B. et al. Correction: GIP receptor agonism improves dyslipidemia and atherosclerosis independently of body weight loss in preclinical mouse model for cardio-metabolic disease. Cardiovasc Diabetol23, 341 (2024). https://doi.org/10.1186/s12933-024-02407-8
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