激活铁突变驱动轴的合成载体

IF 14.7 1区 综合性期刊 Q1 MULTIDISCIPLINARY SCIENCES
Jun Jiang, Lili Yang, Qianqian Xie, Xi Liu, Jie Jiang, Jie Zhang, Shuping Zhang, Huizhen Zheng, Wenjie Li, Xiaoming Cai, Sijin Liu, Ruibin Li
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引用次数: 0

摘要

铁突变是一种很有前景的癌症治疗策略,目前有许多抑制铁突变制动轴的潜在药物正在接受研究。然而,很少有研究探讨激活驱动轴以诱导铁突变的潜力。本文合成了过氧化磷脂酰胆碱装饰脂质体(LIPPCPO),通过靶向二价金属转运体1(DMT1)诱导铁变态反应。研究发现,LIPPCPO 可通过诱导溶酶体 DMT1 的半胱氨酰化来促进溶酶体 Fe2+ 外流,从而抑制谷胱甘肽过氧化物酶 4 (GPX4)、消耗谷胱甘肽并导致乳腺癌细胞和异种移植物发生铁沉降。重要的是,LIPPCPO 诱导的铁氧化细胞死亡与 11 种癌细胞株对放疗、化疗或靶向药物的获得性抗性无关。此外,在 LIPPCPO 和一种美国 FDA 批准的药物青蒿琥酯以及 X 射线之间还观察到了强烈的协同铁变态反应效应。包裹青蒿琥酯的 LIPPCPO 配方能显著抑制肿瘤的生长和转移,并提高乳腺癌雌性小鼠的存活率。这些发现为诱导铁变态反应提供了一种独特的策略,并凸显了 LIPPCPO 作为载体协同传统铁变态反应诱导剂治疗效果的潜力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Synthetic vectors for activating the driving axis of ferroptosis

Synthetic vectors for activating the driving axis of ferroptosis

Ferroptosis is a promising strategy for cancer therapy, with numerous inhibitors of its braking axes under investigation as potential drugs. However, few studies have explored the potential of activating the driving axes to induce ferroptosis. Herein, phosphatidylcholine peroxide decorating liposomes (LIPPCPO) are synthesized to induce ferroptosis by targeting divalent metal transporter 1 (DMT1). LIPPCPO is found to boost lysosomal Fe2+ efflux by inducing cysteinylation of lysosomal DMT1, resulting in glutathione peroxidase 4 (GPX4) suppression, glutathione depletion and ferroptosis in breast cancer cells and xenografts. Importantly, LIPPCPO induced ferroptotic cell death is independent of acquired resistance to radiation, chemotherapy, or targeted agents in 11 cancer cell lines. Furthermore, a strong synergistic ferroptosis effect is observed between LIPPCPO and an FDA-approved drug, artesunate, as well as X rays. The formula of LIPPCPO encapsulating artesunate significantly inhibits tumor growth and metastasis and improves the survival rate of breast cancer-bearing female mice. These findings provide a distinct strategy for inducing ferroptosis and highlight the potential of LIPPCPO as a vector to synergize the therapeutic effects of conventional ferroptosis inducers.

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来源期刊
Nature Communications
Nature Communications Biological Science Disciplines-
CiteScore
24.90
自引率
2.40%
发文量
6928
审稿时长
3.7 months
期刊介绍: Nature Communications, an open-access journal, publishes high-quality research spanning all areas of the natural sciences. Papers featured in the journal showcase significant advances relevant to specialists in each respective field. With a 2-year impact factor of 16.6 (2022) and a median time of 8 days from submission to the first editorial decision, Nature Communications is committed to rapid dissemination of research findings. As a multidisciplinary journal, it welcomes contributions from biological, health, physical, chemical, Earth, social, mathematical, applied, and engineering sciences, aiming to highlight important breakthroughs within each domain.
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