装载 STAT6 siRNA 的超声介导纳米气泡通过克服 M2-TAMs 极化抑制 LUSC 细胞中的 TGF-β1-EMT 轴。

IF 4.3 3区 医学 Q1 PHARMACOLOGY & PHARMACY
Hong Shu , Zhi-jian Ren , Hui Li , Yao Zhang , Ci Yin , Fang Nie
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引用次数: 0

摘要

M2样肿瘤相关巨噬细胞(M2-TAMs)与肺鳞癌(LUSC)的转移和不良临床预后密切相关。以往的研究表明,STAT6是参与M2-TAMs极化的重要信号分子,EMT是TAMs促进肿瘤进展的主要途径。然而,人们很少关注 STAT6 抑制对 LUSC 的影响,传统的基因转染方法也很难在免疫细胞中达到理想的基因沉默效果。在此,我们研究了 12-肉豆蔻 13-乙酸酯(PMA)、脂多糖(LPS)诱导 THP-1 成 M1-TAMs 和 M2-TAMs 的最佳浓度。pSTAT6 和 STAT6 在三种类型巨噬细胞中的表达得到了证实,并证明 pSTAT6 可作为 THP-1 衍生的 M2-TAMs 的特异性靶点。超声介导的纳米气泡破坏(UMND)是一种无创、安全的基因递送技术。我们还合成了PLGA-PEI纳米气泡(NBs),通过UMND将STAT6小干扰RNA(siRNA)载入并递送到M2-TAMs中。结果表明,NBs能有效装载siRNA,并具有良好的生物相容性。我们发现,UMND提高了siRNA的转染效率,也增强了pSTAT6的沉默效果和对M2-TAMs的抑制作用。同时,当 STAT6 siRNA 通过 UMND 进入 M2-TAMs 时,可通过转化生长因子-β1(TGF-β1)途径抑制 LUSC 细胞的增殖、迁移、侵袭和 EMT。因此,我们的研究结果证实,UMND是一种理想的siRNA递送策略,揭示了其抑制M2-TAMs极化并最终治疗LUSC的潜力。
本文章由计算机程序翻译,如有差异,请以英文原文为准。

Ultrasound-mediated nanobubbles loaded with STAT6 siRNA inhibit TGF-β1-EMT axis in LUSC cells via overcoming the polarization of M2-TAMs

Ultrasound-mediated nanobubbles loaded with STAT6 siRNA inhibit TGF-β1-EMT axis in LUSC cells via overcoming the polarization of M2-TAMs

M2-like tumor-associated macrophages (M2-TAMs) are closely correlated with metastasis and poor clinical outcomes in lung squamous cell carcinoma (LUSC). Previous studies have demonstrated that STAT6 is an important signaling molecule involved in the polarization of M2-TAMs, EMT is the main way for TAMs to promote tumor progression. However, little attention has been paid to the effect of STAT6 inhibition on LUSC, and it is difficult to achieve an ideal gene silencing effect in immune cells using traditional gene transfection methods. Here, we investigated the optimal concentration of 12-myristic 13-acetate (PMA), lipopolysaccharide (LPS) for the induction of THP-1 into M1-TAMs and M2-TAMs. The expression of pSTAT6 and STAT6 was confirmed in three types of macrophages, and it was demonstrated that pSTAT6 can be used as a specific target of M2-TAMs derived from THP-1. Ultrasound-mediated nanobubble destruction (UMND) is a non-invasive and safe gene delivery technology. We also synthesized PLGA-PEI nanobubbles (NBs) to load and deliver STAT6 small interfering RNA (siRNA) into M2-TAMs via UMND. The results show that the NBs could effectively load with siRNA and had good biocompatibility. We found that UMND enhanced the transfection efficiency of siRNA, as well as the silencing effect of pSTAT6 and the inhibition of M2-TAMs. Simultaneously, when STAT6 siRNA entered M2-TAMs by UMND, proliferation, migration, invasion and EMT in LUSC cells could be inhibited via the transforming growth factor-β1 (TGF-β1) pathway. Therefore, our results confirm that UMND is an ideal siRNA delivery strategy, revealing its potential to inhibit M2-TAMs polarization and ultimately treat LUSC.

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来源期刊
CiteScore
9.60
自引率
2.20%
发文量
248
审稿时长
50 days
期刊介绍: The journal publishes research articles, review articles and scientific commentaries on all aspects of the pharmaceutical sciences with emphasis on conceptual novelty and scientific quality. The Editors welcome articles in this multidisciplinary field, with a focus on topics relevant for drug discovery and development. More specifically, the Journal publishes reports on medicinal chemistry, pharmacology, drug absorption and metabolism, pharmacokinetics and pharmacodynamics, pharmaceutical and biomedical analysis, drug delivery (including gene delivery), drug targeting, pharmaceutical technology, pharmaceutical biotechnology and clinical drug evaluation. The journal will typically not give priority to manuscripts focusing primarily on organic synthesis, natural products, adaptation of analytical approaches, or discussions pertaining to drug policy making. Scientific commentaries and review articles are generally by invitation only or by consent of the Editors. Proceedings of scientific meetings may be published as special issues or supplements to the Journal.
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