回顾:癫痫发作相关巩固和癫痫网络理论。

Frontiers in network physiology Pub Date : 2024-08-22 eCollection Date: 2024-01-01 DOI:10.3389/fnetp.2024.1430934
Mark R Bower
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引用次数: 0

摘要

癫痫是一种复杂的、多方面的疾病,除了癫痫发作外,它还在多个方面对患者造成影响,包括心理、社交和生活质量问题,但众所周知,癫痫还会与睡眠产生相互作用。癫痫发作常常发生在睡眠与觉醒的交界处,癫痫患者常常经历睡眠中断,发作间期痫样放电率在非快速眼动睡眠期间会增加。癫痫网络理论并没有提到睡眠的作用,但最近对癫痫与睡眠之间相互作用的强调表明,癫痫发作后的睡眠也可能参与到癫痫发作的过程中,并通过与长期记忆形成相关的过程共同作用,使癫痫发作随着时间的推移而变得更加严重("癫痫发生")。虽然人们普遍认为癫痫的反复发作源于神经回路功能的异常,但也有可能是睡眠期间神经回路的正常生理功能在病理信号的驱动下帮助了癫痫的发展。在自发癫痫发作前记录多个单个神经元的研究表明,在癫痫发作开始前激活的神经集合在癫痫发作后的睡眠中被重新激活,这与行为神经集合的重新激活类似,被认为参与了长期记忆的形成,这一过程被称为记忆巩固(Memory Consolidation)。因此,睡眠期间癫痫发作相关神经组合的重新激活被描述为癫痫发作相关巩固(SRC)的一个组成部分。这些结果进一步表明,SRC 可被视为癫痫的一个与网络相关的方面,即使在那些神经解剖学起源受限的癫痫发作中也是如此。正如癫痫网络理论所建议的那样,作为干扰癫痫发生的一种手段,干扰 SRC 的疗法可能会提供一些抗致痫治疗益处,即使这种干扰针对的结构原本并不涉及癫痫发作。在此,我们通过概述记忆巩固(Memory Consolidation)、被认为是MC基础的机制、它们与癫痫发作相关巩固的关系,并提出了针对癫痫发作后在发作后睡眠期间干扰癫痫网络激活的新型抗致痫性疗法,展示了如何将癫痫网络理论扩展到与学习相关的神经可塑性机制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Review: seizure-related consolidation and the network theory of epilepsy.

Epilepsy is a complex, multifaceted disease that affects patients in several ways in addition to seizures, including psychological, social, and quality of life issues, but epilepsy is also known to interact with sleep. Seizures often occur at the boundary between sleep and wake, patients with epilepsy often experience disrupted sleep, and the rate of inter-ictal epileptiform discharges increases during non-REM sleep. The Network Theory of Epilepsy did not address a role for sleep, but recent emphasis on the interaction between epilepsy and sleep suggests that post-seizure sleep may also be involved in the process by which seizures arise and become more severe with time ("epileptogenesis") by co-opting processes related to the formation of long-term memories. While it is generally acknowledged that recurrent seizures arise from the aberrant function of neural circuits, it is possible that the progression of epilepsy is aided by normal, physiological function of neural circuits during sleep that are driven by pathological signals. Studies recording multiple, single neurons prior to spontaneous seizures have shown that neural assemblies activated prior to the start of seizures were reactivated during post-seizure sleep, similar to the reactivation of behavioral neural assemblies, which is thought to be involved in the formation of long-term memories, a process known as Memory Consolidation. The reactivation of seizure-related neural assemblies during sleep was thus described as being a component of Seizure-Related Consolidation (SRC). These results further suggest that SRC may viewed as a network-related aspect of epilepsy, even in those seizures that have anatomically restricted neuroanatomical origins. As suggested by the Network Theory of Epilepsy as a means of interfering with ictogenesis, therapies that interfered with SRC may provide some anti-epileptogenic therapeutic benefit, even if the interference targeted structures that were not involved originally in the seizure. Here, we show how the Network Theory of Epilepsy can be expanded to include neural plasticity mechanisms associated with learning by providing an overview of Memory Consolidation, the mechanisms thought to underlie MC, their relation to Seizure-Related Consolidation, and suggesting novel, anti-epileptogenic therapies targeting interference with network activation in epilepsy following seizures during post-seizure sleep.

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