{"title":"更正 \"过表达的 Hsp70 部分通过 PI3K/Akt 信号通路缓解甲醛诱导的人支气管上皮细胞凋亡\"。","authors":"","doi":"10.1002/tox.24401","DOIUrl":null,"url":null,"abstract":"<p>L. Liu, Y. Huang, X. Feng, J. Chen, and Y. Duan, “Overexpressed Hsp70 Alleviated Formaldehyde-Induced Apoptosis Partly via PI3K/Akt Signaling Pathway in Human Bronchial Epithelial Cells,” <i>Environmental Toxicology</i> 34, no. 4 (2019): 495–504, https://doi.org/10.1002/tox.22703.</p><p>FIGURE 2The protein expression level of Hsp70 in HBE-Hsp70 and HBE-NC exposed to formaldehyde. The results were expressed as mean ± SD from three independent experiments, *<i>p</i> < 0.05, compared with the control group; #<i>p</i> < 0.05, compared with HBE-NC treated with the same concentration of formaldehyde.</p><p>FIGURE 5Effect of LY 294002 on formaldehyde-induced apoptosis in HBE-NC and HBE-Hsp70 cells. (A) Graphs obtained by flow cytometry after Annexin V-APC/7AAD dual straining. (B) Apoptosis rates were expressed as mean ± SD (<i>n</i> = 3), *<i>p</i> < 0.05, **<i>p</i> < 0.01, compared with the control group (0 μmol/L formaldehyde); #<i>p</i> < 0.05, ##<i>p</i> < 0.01, compared with HBE-NC + DMSO treated with the same concentration of formaldehyde; +<i>p</i> < 0.05, ++<i>p</i> < 0.01, compared with HBE-Hsp70 + LY294002 treated with the same concentration of formaldehyde.</p><p>We apologize for this error.</p>","PeriodicalId":11756,"journal":{"name":"Environmental Toxicology","volume":"40 1","pages":"154-155"},"PeriodicalIF":4.4000,"publicationDate":"2024-09-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://onlinelibrary.wiley.com/doi/epdf/10.1002/tox.24401","citationCount":"0","resultStr":"{\"title\":\"Correction to “Overexpressed Hsp70 Alleviated Formaldehyde-Induced Apoptosis Partly via PI3K/Akt Signaling Pathway in Human Bronchial Epithelial Cells”\",\"authors\":\"\",\"doi\":\"10.1002/tox.24401\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p>L. Liu, Y. Huang, X. Feng, J. Chen, and Y. Duan, “Overexpressed Hsp70 Alleviated Formaldehyde-Induced Apoptosis Partly via PI3K/Akt Signaling Pathway in Human Bronchial Epithelial Cells,” <i>Environmental Toxicology</i> 34, no. 4 (2019): 495–504, https://doi.org/10.1002/tox.22703.</p><p>FIGURE 2The protein expression level of Hsp70 in HBE-Hsp70 and HBE-NC exposed to formaldehyde. The results were expressed as mean ± SD from three independent experiments, *<i>p</i> < 0.05, compared with the control group; #<i>p</i> < 0.05, compared with HBE-NC treated with the same concentration of formaldehyde.</p><p>FIGURE 5Effect of LY 294002 on formaldehyde-induced apoptosis in HBE-NC and HBE-Hsp70 cells. (A) Graphs obtained by flow cytometry after Annexin V-APC/7AAD dual straining. (B) Apoptosis rates were expressed as mean ± SD (<i>n</i> = 3), *<i>p</i> < 0.05, **<i>p</i> < 0.01, compared with the control group (0 μmol/L formaldehyde); #<i>p</i> < 0.05, ##<i>p</i> < 0.01, compared with HBE-NC + DMSO treated with the same concentration of formaldehyde; +<i>p</i> < 0.05, ++<i>p</i> < 0.01, compared with HBE-Hsp70 + LY294002 treated with the same concentration of formaldehyde.</p><p>We apologize for this error.</p>\",\"PeriodicalId\":11756,\"journal\":{\"name\":\"Environmental Toxicology\",\"volume\":\"40 1\",\"pages\":\"154-155\"},\"PeriodicalIF\":4.4000,\"publicationDate\":\"2024-09-05\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"https://onlinelibrary.wiley.com/doi/epdf/10.1002/tox.24401\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Environmental Toxicology\",\"FirstCategoryId\":\"3\",\"ListUrlMain\":\"https://onlinelibrary.wiley.com/doi/10.1002/tox.24401\",\"RegionNum\":3,\"RegionCategory\":\"医学\",\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"Q2\",\"JCRName\":\"ENVIRONMENTAL SCIENCES\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Environmental Toxicology","FirstCategoryId":"3","ListUrlMain":"https://onlinelibrary.wiley.com/doi/10.1002/tox.24401","RegionNum":3,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"Q2","JCRName":"ENVIRONMENTAL SCIENCES","Score":null,"Total":0}
Correction to “Overexpressed Hsp70 Alleviated Formaldehyde-Induced Apoptosis Partly via PI3K/Akt Signaling Pathway in Human Bronchial Epithelial Cells”
L. Liu, Y. Huang, X. Feng, J. Chen, and Y. Duan, “Overexpressed Hsp70 Alleviated Formaldehyde-Induced Apoptosis Partly via PI3K/Akt Signaling Pathway in Human Bronchial Epithelial Cells,” Environmental Toxicology 34, no. 4 (2019): 495–504, https://doi.org/10.1002/tox.22703.
FIGURE 2The protein expression level of Hsp70 in HBE-Hsp70 and HBE-NC exposed to formaldehyde. The results were expressed as mean ± SD from three independent experiments, *p < 0.05, compared with the control group; #p < 0.05, compared with HBE-NC treated with the same concentration of formaldehyde.
FIGURE 5Effect of LY 294002 on formaldehyde-induced apoptosis in HBE-NC and HBE-Hsp70 cells. (A) Graphs obtained by flow cytometry after Annexin V-APC/7AAD dual straining. (B) Apoptosis rates were expressed as mean ± SD (n = 3), *p < 0.05, **p < 0.01, compared with the control group (0 μmol/L formaldehyde); #p < 0.05, ##p < 0.01, compared with HBE-NC + DMSO treated with the same concentration of formaldehyde; +p < 0.05, ++p < 0.01, compared with HBE-Hsp70 + LY294002 treated with the same concentration of formaldehyde.
期刊介绍：
The journal publishes in the areas of toxicity and toxicology of environmental pollutants in air, dust, sediment, soil and water, and natural toxins in the environment.Of particular interest are:
Toxic or biologically disruptive impacts of anthropogenic chemicals such as pharmaceuticals, industrial organics, agricultural chemicals, and by-products such as chlorinated compounds from water disinfection and waste incineration;
Natural toxins and their impacts;
Biotransformation and metabolism of toxigenic compounds, food chains for toxin accumulation or biodegradation;
Assays of toxicity, endocrine disruption, mutagenicity, carcinogenicity, ecosystem impact and health hazard;
Environmental and public health risk assessment, environmental guidelines, environmental policy for toxicants.
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