p53 可确保细胞核中 G1/S 特异性组蛋白 H3.1 的正常行为和修饰。

IF 3.3 2区 生物学 Q1 BIOLOGY
Life Science Alliance Pub Date : 2024-06-21 Print Date: 2024-09-01 DOI:10.26508/lsa.202402835
Tsukasa Oikawa, Junya Hasegawa, Haruka Handa, Naomi Ohnishi, Yasuhito Onodera, Ari Hashimoto, Junko Sasaki, Takehiko Sasaki, Koji Ueda, Hisataka Sabe
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引用次数: 0

摘要

H3.1组蛋白主要在细胞周期的G1/S期合成并进入细胞核,是复制核小体的新成分。在这里,我们发现 p53 是确保 G1/S 期 H3.1 在细胞核中正常行为和修饰的必要条件,其中 p53 增加了 H3.1 相互作用组中 C-terminal domain nuclear envelope phosphatase 1 (CTDNEP1) 的水平,降低了增强子泽斯特同源物 2 (EZH2) 的水平。在没有 p53 的情况下,H3.1 分子倾向于被束缚在核包膜(NE)或其附近,在那里它们主要被 EZH2 在赖氨酸 27(H3K27me3)处三甲基化,而不形成核小体。p53 通过提高 CTDNEP1 的水平来降低核 PA 水平,CTDNEP1 可激活脂蛋白将 PA 转化为二酰基甘油。此外,我们还发现细胞质 H3 合子 HSC70 可减弱 H3.1-PA 的相互作用,而且我们的分子成像分析表明,H3.1 可能在进入核后锚定在 NE 周围。我们的研究结果拓展了我们对 p53 在 G1/S 期调控 H3.1 核行为功能的认识,在这一阶段,p53 可能主要以核 PA 和 EZH2 为目标。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
p53 ensures the normal behavior and modification of G1/S-specific histone H3.1 in the nucleus.

H3.1 histone is predominantly synthesized and enters the nucleus during the G1/S phase of the cell cycle, as a new component of duplicating nucleosomes. Here, we found that p53 is necessary to secure the normal behavior and modification of H3.1 in the nucleus during the G1/S phase, in which p53 increases C-terminal domain nuclear envelope phosphatase 1 (CTDNEP1) levels and decreases enhancer of zeste homolog 2 (EZH2) levels in the H3.1 interactome. In the absence of p53, H3.1 molecules tended to be tethered at or near the nuclear envelope (NE), where they were predominantly trimethylated at lysine 27 (H3K27me3) by EZH2, without forming nucleosomes. This accumulation was likely caused by the high affinity of H3.1 toward phosphatidic acid (PA). p53 reduced nuclear PA levels by increasing levels of CTDNEP1, which activates lipin to convert PA into diacylglycerol. We moreover found that the cytosolic H3 chaperone HSC70 attenuates the H3.1-PA interaction, and our molecular imaging analyses suggested that H3.1 may be anchored around the NE after their nuclear entry. Our results expand our knowledge of p53 function in regulation of the nuclear behavior of H3.1 during the G1/S phase, in which p53 may primarily target nuclear PA and EZH2.

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来源期刊
Life Science Alliance
Life Science Alliance Agricultural and Biological Sciences-Plant Science
CiteScore
5.80
自引率
2.30%
发文量
241
审稿时长
10 weeks
期刊介绍: Life Science Alliance is a global, open-access, editorially independent, and peer-reviewed journal launched by an alliance of EMBO Press, Rockefeller University Press, and Cold Spring Harbor Laboratory Press. Life Science Alliance is committed to rapid, fair, and transparent publication of valuable research from across all areas in the life sciences.
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