[撤稿】Mst1 通过 ROCK1/F-actin 途径诱导线粒体损伤,从而调控非小细胞肺癌 A549 细胞凋亡。

IF 4.5 3区 医学 Q1 ONCOLOGY
International journal of oncology Pub Date : 2024-08-01 Epub Date: 2024-06-14 DOI:10.3892/ijo.2024.5663
Weiqiang Zhang, Keiqiang Liu, Yingxin Pei, Jingbo Ma, Jiang Tan, Jing Zhao
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引用次数: 0

摘要

在上述文章发表后,一位感兴趣的读者提请作者注意,第 2418 页图 7E 中显示的某些 EdU 检测数据已经以不同的形式出现在以前发表的一篇论文中,该论文由不同的作者在不同的研究机构撰写。由于上述文章中有争议的数据在提交给《国际肿瘤学杂志》之前已经发表,因此编辑决定从该杂志上撤下这篇论文。已要求作者就这些问题做出解释,但编辑部没有收到回复。对于给读者带来的不便,编辑深表歉意。 [International Journal of Oncology 53: 2409-2422, 2018; DOI: 10.3892/ijo.2018.4586]。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
[Retracted] Mst1 regulates non‑small cell lung cancer A549 cell apoptosis by inducing mitochondrial damage via ROCK1/F‑actin pathways.

Subsequently to the publication of the above article, an interested reader drew to the authors' attention that certain of the EdU assay data shown in Fig. 7E on p. 2418 had already appeared in different form in a previously published paper written by different authors at different research institutes. Owing to the fact that the contentious data in the above article had already been published prior to its submission to International Journal of Oncology, the Editor has decided that this paper should be retracted from the Journal. The authors were asked for an explanation to account for these concerns, but the Editorial Office did not receive a reply. The Editor apologizes to the readership for any inconvenience caused.  [International Journal of Oncology 53: 2409‑2422, 2018; DOI: 10.3892/ijo.2018.4586].

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来源期刊
CiteScore
9.60
自引率
0.00%
发文量
157
审稿时长
2.1 months
期刊介绍: The main aim of Spandidos Publications is to facilitate scientific communication in a clear, concise and objective manner, while striving to provide prompt publication of original works of high quality. The journals largely concentrate on molecular and experimental medicine, oncology, clinical and experimental cancer treatment and biomedical research. All journals published by Spandidos Publications Ltd. maintain the highest standards of quality, and the members of their Editorial Boards are world-renowned scientists.
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