撤回。

IF 3 3区 生物学 Q3 BIOCHEMISTRY & MOLECULAR BIOLOGY
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引用次数: 0

摘要

撤回:"(2R,3R)二氢杨梅素通过清除LPS诱导的氧化应激及NF-κB和MAPKs通路激活抑制破骨细胞生成和骨丢失",作者:张学军,李鑫,方建国,侯小龙,黄芳,郭凤静,李锋,陈安民,黄世龙,J Cell Biochem 2018, 119: 8981-8995。上述文章于2018年8月4日在线发表于《Wiley Online Library》(https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.27154),经该刊主编Christian Behl和Wiley Periodicals LLC协议,该文章已被撤回。撤稿协议是根据第三方提出的指控进行调查后达成的。在调查过程中,发现所提供的数据存在若干缺陷和不一致之处。因此,编辑认为这篇文章的结论无效。作者已收到撤稿通知,但无法得到最终确认。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Retraction

Retraction: “(2R,3R)Dihydromyricetin inhibits osteoclastogenesis and bone loss through scavenging LPS-induced oxidative stress and NF-κB and MAPKs pathways activating,” by Xuejun Zhang, Xin Li, Jianguo Fang, Xiaolong Hou, Huang Fang, Fengjing Guo, Feng Li, Anmin Chen, Shilong Huang, J Cell Biochem 2018, 119: 8981-8995. The above article, published online on 4 August 2018 in Wiley Online Library (https://onlinelibrary.wiley.com/doi/full/10.1002/jcb.27154) has been retracted by agreement between the journal's Editor in Chief, Christian Behl, and Wiley Periodicals LLC.

The retraction has been agreed following an investigation based on allegations raised by third parties. During the investigation, several flaws and inconsistencies were found within the data presented. Thus, the editors consider the conclusions of this article to be invalid. The authors have been informed of the retraction but were not available for a final confirmation.

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来源期刊
Journal of cellular biochemistry
Journal of cellular biochemistry 生物-生化与分子生物学
CiteScore
9.90
自引率
0.00%
发文量
164
审稿时长
1 months
期刊介绍: The Journal of Cellular Biochemistry publishes descriptions of original research in which complex cellular, pathogenic, clinical, or animal model systems are studied by biochemical, molecular, genetic, epigenetic or quantitative ultrastructural approaches. Submission of papers reporting genomic, proteomic, bioinformatics and systems biology approaches to identify and characterize parameters of biological control in a cellular context are encouraged. The areas covered include, but are not restricted to, conditions, agents, regulatory networks, or differentiation states that influence structure, cell cycle & growth control, structure-function relationships.
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