MiR-29和MiR-140调控TRAIL诱导的肺癌药物耐受性

IF 4.6 Q2 MATERIALS SCIENCE, BIOMATERIALS
Suyeon Kim, Ki Wook Lee, Yongjin Yoo, Sang Hee Park, Ji Won Lee, Suhyun Jeon, Shaginyan Illia, Pooja Joshi, Hyun Woo Park, Han-En Lo, Jimin Seo, Yeonwoo Kim, Min Chang, Tae Jin Lee, Jong Bae Seo, Sung-Hak Kim, Carlo M. Croce, Inki Kim, Sung-Suk Suh, Young-Jun Jeon
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引用次数: 0

摘要

肿瘤坏死因子相关凋亡诱导配体(TRAIL)作为一种外源性凋亡配体的调节剂,具有化疗潜力,但其作为药物的效果受到先天和后天因素的限制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
MiR-29 and MiR-140 regulate TRAIL-induced drug tolerance in lung cancer
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) has chemotherapeutic potential as a regulator of an extrinsic apoptotic ligand, but its effect as a drug is limited by innate and acq...
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来源期刊
ACS Applied Bio Materials
ACS Applied Bio Materials Chemistry-Chemistry (all)
CiteScore
9.40
自引率
2.10%
发文量
464
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