MiR-29和MiR-140调控TRAIL诱导的肺癌药物耐受性

IF 2.5 2区 生物学 Q3 CELL BIOLOGY
Suyeon Kim, Ki Wook Lee, Yongjin Yoo, Sang Hee Park, Ji Won Lee, Suhyun Jeon, Shaginyan Illia, Pooja Joshi, Hyun Woo Park, Han-En Lo, Jimin Seo, Yeonwoo Kim, Min Chang, Tae Jin Lee, Jong Bae Seo, Sung-Hak Kim, Carlo M. Croce, Inki Kim, Sung-Suk Suh, Young-Jun Jeon
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引用次数: 0

摘要

肿瘤坏死因子相关凋亡诱导配体(TRAIL)作为一种外源性凋亡配体的调节剂,具有化疗潜力,但其作为药物的效果受到先天和后天因素的限制。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
MiR-29 and MiR-140 regulate TRAIL-induced drug tolerance in lung cancer
Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) has chemotherapeutic potential as a regulator of an extrinsic apoptotic ligand, but its effect as a drug is limited by innate and acq...
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来源期刊
Animal Cells and Systems
Animal Cells and Systems 生物-动物学
CiteScore
4.50
自引率
24.10%
发文量
33
审稿时长
6 months
期刊介绍: Animal Cells and Systems is the official journal of the Korean Society for Integrative Biology. This international, peer-reviewed journal publishes original papers that cover diverse aspects of biological sciences including Bioinformatics and Systems Biology, Developmental Biology, Evolution and Systematic Biology, Population Biology, & Animal Behaviour, Molecular and Cellular Biology, Neurobiology and Immunology, and Translational Medicine.
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