躯体症状失调的心理行为 B 级标准与大样本心身疾病门诊患者的躯体症状报告有关

Matthias Hoheisel, Stoyan Popkirov, Rosa Michaelis, Matthias Rose
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引用次数: 0

摘要

导言:DSM-5 引入的躯体症状障碍(SSD)的特点是慢性躯体症状无法完全由潜在病理解释,并伴有心理因素,即诊断 B 标准。这些认知、情感和行为障碍与对躯体症状的关注增加有关。然而,在临床环境中,B标准与高症状报告之间的关系还缺乏实证证据:这项为期 12 年的回顾性、横断面观察研究调查了一家德国心身疾病门诊中心的 6 491 名患者。健康-49》的躯体形式分量表用于评估躯体症状报告。使用 ICD-10 症状分级和 HEALTH-49 的其他分量表确定了与症状报告相关的过度健康关注和其他潜在标准。结果显示回归分析表明,已确立的 SSD B 标准是与躯体症状报告相关的最强因素,其标准化贝塔系数为 β = 0.31(R² = .428,df = 24,F = 187.886)。抑郁症状以及活动和参与能力受损与躯体症状报告的相关性显然较低。社会人口学因素,如年龄(β = 0.16)和性别(β = 0.12),也与躯体症状报告独立相关:本研究基于大量患者样本,为与躯体症状报告相关的特定 B 标准有关的 SSD 概念提供了证据。这些结果表明了心理症状在躯体症状患者中的重要作用。研究结果还表明,其他因素也会导致躯体症状的报告。我们的研究结果可为未来的躯体疾病诊断标准提供参考。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Psychobehavioural B-criteria of somatic symptom disorder are associated with somatic symptom reporting in a large sample of psychosomatic outpatients
Introduction: Somatic Symptom Disorder (SSD) as introduced by the DSM-5 is characterized by chronic somatic symptoms not fully explained by underlying pathology and accompanied by psychological factors, the diagnostic B-criteria. These cognitive, affective, and behavioral disturbances are related to increased attention to somatic symptoms. However, there is a lack of empirical evidence regarding the association between the B-criteria and high symptom reporting in clinical settings. Methods: This 12-year retrospective, cross-sectional, observational study examined 6,491 patients from a german psychosomatic outpatient center. The somatoform subscale of HEALTH-49 was used to evaluate somatic symptom reporting. Excessive health concerns and other potential criteria associated with symptom reporting were determined using the ICD-10-Symptom Rating and other HEALTH-49 subscales. Results: Regression analysis revealed that the established B-criteria for SSD were the strongest factors associated with somatic symptom reporting, with a standardized beta coefficient of β = 0.31 (R² = .428, df = 24, F = 187.886). Depressive symptoms and impaired activity and participation were clearly less associated with somatic symptom reporting. Sociodemographic factors, such as age (β = 0.16) and gender (β = 0.12), were also independently associated with somatic symptom reporting. Conclusion: This study provides evidence for the concept of SSD related to specific B-criteria associated with somatic symptom reporting, based on a large patient sample. These results point to an important role of psychological symptomatology in patients with somatic symptoms. The findings also suggest that additional factors contribute to the reporting of somatic symptoms. Our results may inform future diagnostic criteria for SSD.
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