增强子突变可调节化疗引起的骨髓抑制的严重程度。

IF 3.3 2区 生物学 Q1 BIOLOGY
Life Science Alliance Pub Date : 2024-01-16 Print Date: 2024-03-01 DOI:10.26508/lsa.202302244
Artemy Zhigulev, Zandra Norberg, Julie Cordier, Rapolas Spalinskas, Hassan Bassereh, Niclas Björn, Sailendra Pradhananga, Henrik Gréen, Pelin Sahlén
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引用次数: 0

摘要

非小细胞肺癌通常在晚期才被确诊,许多患者仍在接受传统化疗。化疗的非选择性往往导致严重的骨髓抑制。以前的研究表明,蛋白编码突变不能完全解释骨髓抑制的易感性。在此,我们研究了增强子突变在骨髓抑制易感性中可能扮演的角色。我们制作了用卡铂或吉西他滨治疗的三个类造血干细胞系的转录组和启动子相互作用图(使用 HiCap)。利用公开的增强子数据集,我们使用表观遗传 CRISPR 技术在硅学和活细胞中验证了 HiCap 的结果。我们还开发了一种网络方法,用于相互作用组分析和差异相互作用基因的检测。与大体水平的差异基因表达分析相比,差异相互作用分析为骨髓抑制的相关基因和通路提供了更多信息。此外,我们还发现,差异相互作用基因的增强子高度富集了与不同程度骨髓抑制相关的变异。总之,我们的工作是综合转录组和基因调控数据集分析非编码突变功能注释的杰出范例。
本文章由计算机程序翻译,如有差异,请以英文原文为准。
Enhancer mutations modulate the severity of chemotherapy-induced myelosuppression.

Non-small cell lung cancer is often diagnosed at advanced stages, and many patients are still treated with classical chemotherapy. The unselective nature of chemotherapy often results in severe myelosuppression. Previous studies showed that protein-coding mutations could not fully explain the predisposition to myelosuppression. Here, we investigate the possible role of enhancer mutations in myelosuppression susceptibility. We produced transcriptome and promoter-interaction maps (using HiCap) of three blood stem-like cell lines treated with carboplatin or gemcitabine. Taking advantage of publicly available enhancer datasets, we validated HiCap results in silico and in living cells using epigenetic CRISPR technology. We also developed a network approach for interactome analysis and detection of differentially interacting genes. Differential interaction analysis provided additional information on relevant genes and pathways for myelosuppression compared with differential gene expression analysis at the bulk level. Moreover, we showed that enhancers of differentially interacting genes are highly enriched for variants associated with differing levels of myelosuppression. Altogether, our work represents a prominent example of integrative transcriptome and gene regulatory datasets analysis for the functional annotation of noncoding mutations.

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来源期刊
Life Science Alliance
Life Science Alliance Agricultural and Biological Sciences-Plant Science
CiteScore
5.80
自引率
2.30%
发文量
241
审稿时长
10 weeks
期刊介绍: Life Science Alliance is a global, open-access, editorially independent, and peer-reviewed journal launched by an alliance of EMBO Press, Rockefeller University Press, and Cold Spring Harbor Laboratory Press. Life Science Alliance is committed to rapid, fair, and transparent publication of valuable research from across all areas in the life sciences.
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