V V Strekalova, D G Khachirov, A N Dedenkov, Iu I Suvorov, I K Shvatsabaia
{"title":"[Wistar大鼠慢性盐负荷联合低蛋白饮食的实验性高血压模型]。","authors":"V V Strekalova, D G Khachirov, A N Dedenkov, Iu I Suvorov, I K Shvatsabaia","doi":"","DOIUrl":null,"url":null,"abstract":"<p><p>Combination of chronic salt loading with protein-poor diet produces experimental hypertension with natrium consumption near to physiological. The present model is characterized, compared to the existing one, by stage development, moderate arterial blood pressure elevation and absence of \"salt toxicosis\" and may be thus considered more adequate for experimental investigation of primary arterial hypertension pathophysiology.</p>","PeriodicalId":77680,"journal":{"name":"Biulleten' Vsesoiuznogo kardiologicheskogo nauchnogo tsentra AMN SSSR","volume":"12 1","pages":"48-51"},"PeriodicalIF":0.0000,"publicationDate":"1989-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":"0","resultStr":"{\"title\":\"[Modeling of experimental hypertension by chronic salt loading combined with a low-protein diet in Wistar rats].\",\"authors\":\"V V Strekalova, D G Khachirov, A N Dedenkov, Iu I Suvorov, I K Shvatsabaia\",\"doi\":\"\",\"DOIUrl\":null,\"url\":null,\"abstract\":\"<p><p>Combination of chronic salt loading with protein-poor diet produces experimental hypertension with natrium consumption near to physiological. The present model is characterized, compared to the existing one, by stage development, moderate arterial blood pressure elevation and absence of \\\"salt toxicosis\\\" and may be thus considered more adequate for experimental investigation of primary arterial hypertension pathophysiology.</p>\",\"PeriodicalId\":77680,\"journal\":{\"name\":\"Biulleten' Vsesoiuznogo kardiologicheskogo nauchnogo tsentra AMN SSSR\",\"volume\":\"12 1\",\"pages\":\"48-51\"},\"PeriodicalIF\":0.0000,\"publicationDate\":\"1989-01-01\",\"publicationTypes\":\"Journal Article\",\"fieldsOfStudy\":null,\"isOpenAccess\":false,\"openAccessPdf\":\"\",\"citationCount\":\"0\",\"resultStr\":null,\"platform\":\"Semanticscholar\",\"paperid\":null,\"PeriodicalName\":\"Biulleten' Vsesoiuznogo kardiologicheskogo nauchnogo tsentra AMN SSSR\",\"FirstCategoryId\":\"1085\",\"ListUrlMain\":\"\",\"RegionNum\":0,\"RegionCategory\":null,\"ArticlePicture\":[],\"TitleCN\":null,\"AbstractTextCN\":null,\"PMCID\":null,\"EPubDate\":\"\",\"PubModel\":\"\",\"JCR\":\"\",\"JCRName\":\"\",\"Score\":null,\"Total\":0}","platform":"Semanticscholar","paperid":null,"PeriodicalName":"Biulleten' Vsesoiuznogo kardiologicheskogo nauchnogo tsentra AMN SSSR","FirstCategoryId":"1085","ListUrlMain":"","RegionNum":0,"RegionCategory":null,"ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":null,"EPubDate":"","PubModel":"","JCR":"","JCRName":"","Score":null,"Total":0}
[Modeling of experimental hypertension by chronic salt loading combined with a low-protein diet in Wistar rats].
Combination of chronic salt loading with protein-poor diet produces experimental hypertension with natrium consumption near to physiological. The present model is characterized, compared to the existing one, by stage development, moderate arterial blood pressure elevation and absence of "salt toxicosis" and may be thus considered more adequate for experimental investigation of primary arterial hypertension pathophysiology.